TMT induces apoptosis and necroptosis in mouse kidneys through oxidative stress-induced activation of the NLRP3 inflammasome
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Title
TMT induces apoptosis and necroptosis in mouse kidneys through oxidative stress-induced activation of the NLRP3 inflammasome
Authors
Keywords
Trimethyltin chloride, Oxidative stress, Inflammasomes, Apoptosis, Necroptosis
Journal
ECOTOXICOLOGY AND ENVIRONMENTAL SAFETY
Volume 230, Issue -, Pages 113167
Publisher
Elsevier BV
Online
2022-01-05
DOI
10.1016/j.ecoenv.2022.113167
References
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- (2021) Jingzeng Cai et al. BIOFACTORS
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- (2021) V Quagliariello et al. EUROPEAN HEART JOURNAL
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- (2021) Asrin Ahmadi et al. TOXICOLOGY LETTERS
- KIF5A-dependent axonal transport deficiency disrupts autophagic flux in trimethyltin chloride-induced neurotoxicity
- (2020) Mengyu Liu et al. Autophagy
- Environmental contaminant ammonia triggers epithelial-to-mesenchymal transition-mediated jejunal fibrosis with the disassembly of epithelial cell-cell contacts in chicken
- (2020) Hongyuan Jing et al. SCIENCE OF THE TOTAL ENVIRONMENT
- H2S exposure-induced oxidative stress promotes LPS-mediated hepatocyte autophagy through the PI3K/AKT/TOR pathway
- (2020) Jin-ming Guo et al. ECOTOXICOLOGY AND ENVIRONMENTAL SAFETY
- Ammonia regulates chicken tracheal cell necroptosis via the LncRNA-107053293/MiR-148a-3p/FAF1 axis
- (2019) Wei Wang et al. JOURNAL OF HAZARDOUS MATERIALS
- Ethyl Acetate Fraction from Persimmon (Diospyros kaki) Ameliorates Cerebral Neuronal Loss and Cognitive Deficit via the JNK/Akt Pathway in TMT-Induced Mice
- (2018) Jong Kim et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
- Mitochondrial network structure homeostasis and cell death
- (2018) Long-long Xie et al. CANCER SCIENCE
- Trimethyltin chloride induces reactive oxygen species-mediated apoptosis in retinal cells during zebrafish eye development
- (2018) Jin Kim et al. SCIENCE OF THE TOTAL ENVIRONMENT
- Autophagy blockade and lysosomal membrane permeabilization contribute to lead-induced nephrotoxicity in primary rat proximal tubular cells
- (2017) Xiang-Bin Song et al. Cell Death & Disease
- Lycium barbarumPolysaccharides Protect against Trimethyltin Chloride-Induced Apoptosis via Sonic Hedgehog and PI3K/Akt Signaling Pathways in Mouse Neuro-2a Cells
- (2016) Wanyun Zhao et al. Oxidative Medicine and Cellular Longevity
- NLRP3 inflammasome mediates contrast media-induced acute kidney injury by regulating cell apoptosis
- (2016) Jianxiao Shen et al. Scientific Reports
- Caspase-8 as an Effector and Regulator of NLRP3 Inflammasome Signaling
- (2015) Christina Antonopoulos et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Chronic trimethyltin chloride exposure and the development of kidney stones in rats
- (2014) Xuefeng Ren et al. JOURNAL OF APPLIED TOXICOLOGY
- Caspase-8 and RIP kinases regulate bacteria-induced innate immune responses and cell death
- (2014) D. Weng et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Apoptosis induced by Trimethyltin chloride in human neuroblastoma cells SY5Y is regulated by a balance and cross-talk between NF-κB and MAPKs signaling pathways
- (2013) Yan Qing et al. ARCHIVES OF TOXICOLOGY
- Organotin Compounds, Including Butyltins and Octyltins, in House Dust from Albany, New York, USA
- (2010) Kurunthachalam Kannan et al. ARCHIVES OF ENVIRONMENTAL CONTAMINATION AND TOXICOLOGY
- The NLRP3 Inflammasome: A Sensor for Metabolic Danger?
- (2010) K. Schroder et al. SCIENCE
- Mechanism underlying hypokalemia induced by trimethyltin chloride: Inhibition of H+/K+-ATPase in renal intercalated cells
- (2010) Xiaojiang Tang et al. TOXICOLOGY
- The concentration-dependent induction of cell death by trimethyltin chloride in rat liver epithelial IAR20 cells
- (2008) Mengmeng Wang et al. TOXICOLOGY IN VITRO
- Environmental levels, toxicity and human exposure to tributyltin (TBT)-contaminated marine environment. A review
- (2007) Blanca Antizar-Ladislao ENVIRONMENT INTERNATIONAL
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