Mechanism of mitochondrial permeability transition pore induction and damage in the pancreas: inhibition prevents acute pancreatitis by protecting production of ATP
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Title
Mechanism of mitochondrial permeability transition pore induction and damage in the pancreas: inhibition prevents acute pancreatitis by protecting production of ATP
Authors
Keywords
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Journal
GUT
Volume 65, Issue 8, Pages 1333-1346
Publisher
BMJ
Online
2015-06-13
DOI
10.1136/gutjnl-2014-308553
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Note: Only part of the references are listed.- The mitochondrial permeability transition: A current perspective on its identity and role in ischaemia/reperfusion injury
- (2015) Andrew P. Halestrap et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Cyclophilin inhibition as potential therapy for liver diseases
- (2014) Nikolai V. Naoumov JOURNAL OF HEPATOLOGY
- An uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition pore
- (2014) K. N. Alavian et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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- (2013) Wei Huang et al. GUT
- IL-6 trans-signaling promotes pancreatitis-associated lung injury and lethality
- (2013) Hong Zhang et al. JOURNAL OF CLINICAL INVESTIGATION
- Dimers of mitochondrial ATP synthase form the permeability transition pore
- (2013) V. Giorgio et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Ca2+ release-activated Ca2+ channel blockade as a potential tool in antipancreatitis therapy
- (2013) J. V. Gerasimenko et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Autophagy and pancreatitis
- (2012) Anna S. Gukovskaya et al. AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY
- The Mitochondrial Phosphatase PGAM5 Functions at the Convergence Point of Multiple Necrotic Death Pathways
- (2012) Zhigao Wang et al. CELL
- Preventing Pancreatitis by Protecting the Mitochondrial Permeability Transition Pore
- (2012) Markus M. Lerch et al. GASTROENTEROLOGY
- Effects of Oxidative Alcohol Metabolism on the Mitochondrial Permeability Transition Pore and Necrosis in a Mouse Model of Alcoholic Pancreatitis
- (2012) Natalia Shalbueva et al. GASTROENTEROLOGY
- Rhomboid Protease PARL Mediates the Mitochondrial Membrane Potential Loss-induced Cleavage of PGAM5
- (2012) Shiori Sekine et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Bile Acids Induce Pancreatic Acinar Cell Injury and Pancreatitis by Activating Calcineurin
- (2012) Kamaldeen A. Muili et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Intra-acinar Trypsinogen Activation Mediates Early Stages of Pancreatic Injury but Not Inflammation in Mice With Acute Pancreatitis
- (2011) Rajinder Dawra et al. GASTROENTEROLOGY
- TLR9 and the NLRP3 Inflammasome Link Acinar Cell Death With Inflammation in Acute Pancreatitis
- (2011) Rafaz Hoque et al. GASTROENTEROLOGY
- Reactive Oxygen Species Induced by Bile Acid Induce Apoptosis and Protect Against Necrosis in Pancreatic Acinar Cells
- (2011) David M. Booth et al. GASTROENTEROLOGY
- Intracellular activation of trypsinogen in transgenic mice induces acute but not chronic pancreatitis
- (2011) S. Gaiser et al. GUT
- Lipotoxicity Causes Multisystem Organ Failure and Exacerbates Acute Pancreatitis in Obesity
- (2011) S. Navina et al. Science Translational Medicine
- Dynamic Changes in Cytosolic and Mitochondrial ATP Levels in Pancreatic Acinar Cells
- (2010) Svetlana G. Voronina et al. GASTROENTEROLOGY
- Organ Failure and Infection of Pancreatic Necrosis as Determinants of Mortality in Patients With Acute Pancreatitis
- (2010) Maxim S. Petrov et al. GASTROENTEROLOGY
- Cyclophilin D controls mitochondrial pore–dependent Ca2+ exchange, metabolic flexibility, and propensity for heart failure in mice
- (2010) John W. Elrod et al. JOURNAL OF CLINICAL INVESTIGATION
- TRO40303, a New Cardioprotective Compound, Inhibits Mitochondrial Permeability Transition
- (2010) S. Schaller et al. JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS
- Autophagy proteins regulate innate immune responses by inhibiting the release of mitochondrial DNA mediated by the NALP3 inflammasome
- (2010) Kiichi Nakahira et al. NATURE IMMUNOLOGY
- Molecular mechanisms of necroptosis: an ordered cellular explosion
- (2010) Peter Vandenabeele et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- Regulation and pharmacology of the mitochondrial permeability transition pore
- (2009) D. B. Zorov et al. CARDIOVASCULAR RESEARCH
- Receptor Interacting Protein Kinase-3 Determines Cellular Necrotic Response to TNF-α
- (2009) Sudan He et al. CELL
- Prosurvival Bcl-2 proteins stabilize pancreatic mitochondria and protect against necrosis in experimental pancreatitis
- (2009) Kai-Feng Sung et al. EXPERIMENTAL CELL RESEARCH
- Deletion of TRPC3 in Mice Reduces Store-Operated Ca2+ Influx and the Severity of Acute Pancreatitis
- (2009) Min Seuk Kim et al. GASTROENTEROLOGY
- Pancreatic protease activation by alcohol metabolite depends on Ca2+ release via acid store IP3 receptors
- (2009) J. V. Gerasimenko et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Cell death and proliferation in acute slices and organotypic cultures of mammalian CNS
- (2009) Laura Lossi et al. PROGRESS IN NEUROBIOLOGY
- Direct Activation of Cytosolic Ca2+ Signaling and Enzyme Secretion by Cholecystokinin in Human Pancreatic Acinar Cells
- (2008) John A. Murphy et al. GASTROENTEROLOGY
- Mechanisms regulating cytochrome c release in pancreatic mitochondria
- (2008) I V Odinokova et al. GUT
- Polarized Calcium Signaling in Exocrine Gland Cells
- (2007) Ole H. Petersen et al. Annual Review of Physiology
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