MicroRNA‐590‐3p inhibits T helper 17 cells and ameliorates inflammation in lupus mice

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IL-12 and IL-23/Th17 axis in systemic lupus erythematosus

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miR-590-3p Targets Cyclin G2 and FOXO3 to Promote Ovarian Cancer Cell Proliferation, Invasion, and Spheroid Formation

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LncRNA PART‐1 targets TGFBR2/Smad3 to regulate cell viability and apoptosis of chondrocytes via acting as miR‐590‐3p sponge in osteoarthritis

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MiR‐590‐3p regulates proliferation, migration and collagen synthesis of cardiac fibroblast by targeting ZEB1

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miR-223-3p promotes autoreactive Th17 cell responses in experimental autoimmune uveitis (EAU) by inhibiting transcription factor FOXO3 expression

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Chloroquine inhibits autophagic flux by decreasing autophagosome-lysosome fusion

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MicroRNA-181c promotes Th17 cell differentiation and mediates experimental autoimmune encephalomyelitis

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Loss of PPM1F expression predicts tumour recurrence and is negatively regulated by miR-590-3p in gastric cancer

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Hydroxychloroquine Inhibits the Differentiation of Th17 Cells in Systemic Lupus Erythematosus

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Long Non-coding MIR205HG Depletes Hsa-miR-590-3p Leading to Unrestrained Proliferation in Head and Neck Squamous Cell Carcinoma

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miR-590–3p inhibits proliferation and promotes apoptosis by targeting activating transcription factor 3 in human breast cancer cells

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FAS and RAS related Apoptosis defects: From autoimmunity to leukemia

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MicroRNA-590-3P suppresses cell survival and triggers breast cancer cell apoptosis via targeting sirtuin-1 and deacetylation of p53

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Overexpression of microRNA-590-3p promotes the proliferation of and inhibits the apoptosis of myocardial cells through inhibition of the NF-κB signaling pathway by binding to RIPK1

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Combination of Endothelial-Monocyte-Activating Polypeptide-II with Temozolomide Suppress Malignant Biological Behaviors of Human Glioblastoma Stem Cells via miR-590-3p/MACC1 Inhibiting PI3K/AKT/mTOR Signal Pathway

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Control of lupus nephritis by changes of gut microbiota

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MiR-30a inhibits Th17 differentiation and demyelination of EAE mice by targeting the IL-21R

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Macroautophagy inhibition maintains fragmented mitochondria to foster T cell receptor‐dependent apoptosis

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Increased autophagy in CD4+T cells of rheumatoid arthritis patients results in T-cell hyperactivation and apoptosis resistance

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The MicroRNA-183-96-182 Cluster Promotes T Helper 17 Cell Pathogenicity by Negatively Regulating Transcription Factor Foxo1 Expression

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Autophagy proteins in antigen processing for presentation on MHC molecules

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Treg17 cells are programmed by Stat3 to suppress Th17 responses in systemic lupus

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Oxidative Stress and Treg and Th17 Dysfunction in Systemic Lupus Erythematosus

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A rapid and high content assay that measures cyto-ID-stained autophagic compartments and estimates autophagy flux with potential clinical applications

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MicroRNA-21 promotes Th17 differentiation and mediates experimental autoimmune encephalomyelitis

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miR-10a inhibits dendritic cell activation and Th1/Th17 cell immune responses in IBD

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DNA methylation and mRNA and microRNA expression of SLE CD4+ T cells correlate with disease phenotype

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The Intracellular Sensor NOD2 Induces MicroRNA-29 Expression in Human Dendritic Cells to Limit IL-23 Release

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Clinical associations of serum interleukin-17 in systemic lupus erythematosus

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Autophagy promotes T-cell survival through degradation of proteins of the cell death machinery

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Th17 Cells Induce Ectopic Lymphoid Follicles in Central Nervous System Tissue Inflammation

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Macroautophagy Regulates Energy Metabolism during Effector T Cell Activation

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Proinflammatory T helper type 17 cells are effective B-cell helpers

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Th17 and natural Treg cell population dynamics in systemic lupus erythematosus

(2009) Ji Yang et al.   ARTHRITIS AND RHEUMATISM

Logic and Extent of miRNA-Mediated Control of Autoimmune Gene Expression

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Autophagy Is Essential for Mitochondrial Clearance in Mature T Lymphocytes

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MicroRNAs: new regulators of immune cell development and function

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