Bevacizumab-Induced Mitochondrial Dysfunction, Endoplasmic Reticulum Stress, and ERK Inactivation Contribute to Cardiotoxicity
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Title
Bevacizumab-Induced Mitochondrial Dysfunction, Endoplasmic Reticulum Stress, and ERK Inactivation Contribute to Cardiotoxicity
Authors
Keywords
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Journal
Oxidative Medicine and Cellular Longevity
Volume 2021, Issue -, Pages 1-11
Publisher
Hindawi Limited
Online
2021-03-24
DOI
10.1155/2021/5548130
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