Overexpression of mitochondrial creatine kinase preserves cardiac energetics without ameliorating murine chronic heart failure
Published 2020 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Overexpression of mitochondrial creatine kinase preserves cardiac energetics without ameliorating murine chronic heart failure
Authors
Keywords
-
Journal
BASIC RESEARCH IN CARDIOLOGY
Volume 115, Issue 2, Pages -
Publisher
Springer Science and Business Media LLC
Online
2020-01-10
DOI
10.1007/s00395-020-0777-3
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Over-expression of mitochondrial creatine kinase in the murine heart improves functional recovery and protects against injury following ischaemia–reperfusion
- (2018) Hannah J Whittington et al. CARDIOVASCULAR RESEARCH
- Decreased ATP production and myocardial contractile reserve in metabolic heart disease
- (2018) Ivan Luptak et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Increasing creatine kinase activity protects against hypoxia / reoxygenation injury but not against anthracycline toxicity in vitro
- (2017) Sevasti Zervou et al. PLoS One
- Reversal of Mitochondrial Transhydrogenase Causes Oxidative Stress in Heart Failure
- (2015) Alexander G. Nickel et al. Cell Metabolism
- Attention to Background Strain Is Essential for Metabolic Research: C57BL/6 and the International Knockout Mouse Consortium
- (2015) Danielle A. Fontaine et al. DIABETES
- Synergistic role of ADP and Ca2+in diastolic myocardial stiffness
- (2015) Vasco Sequeira et al. JOURNAL OF PHYSIOLOGY-LONDON
- Metabolic Flux as a Predictor of Heart Failure Prognosis
- (2014) Craig A. Lygate et al. CIRCULATION RESEARCH
- Cardiovascular remodelling in coronary artery disease and heart failure
- (2014) Gerd Heusch et al. LANCET
- Living Without Creatine
- (2013) Craig A. Lygate et al. CIRCULATION RESEARCH
- Ribose Supplementation Alone or with Elevated Creatine Does Not Preserve High Energy Nucleotides or Cardiac Function in the Failing Mouse Heart
- (2013) Kiterie M. E. Faller et al. PLoS One
- Metabolic Rates of ATP Transfer Through Creatine Kinase (CK Flux) Predict Clinical Heart Failure Events and Death
- (2013) P. A. Bottomley et al. Science Translational Medicine
- Chronic creatine kinase deficiency eventually leads to congestive heart failure, but severity is dependent on genetic background, gender and age
- (2012) Craig A. Lygate et al. BASIC RESEARCH IN CARDIOLOGY
- Moderate elevation of intracellular creatine by targeting the creatine transporter protects mice from acute myocardial infarction
- (2012) Craig A. Lygate et al. CARDIOVASCULAR RESEARCH
- Effects of Catecholamine Stress on Diastolic Function and Myocardial Energetics in Obesity
- (2012) Oliver J. Rider et al. CIRCULATION
- Creatine kinase–mediated improvement of function in failing mouse hearts provides causal evidence the failing heart is energy starved
- (2011) Ashish Gupta et al. JOURNAL OF CLINICAL INVESTIGATION
- Impaired ATP Kinetics in Failing In Vivo Mouse Heart
- (2010) Ashish Gupta et al. Circulation-Cardiovascular Imaging
- Abnormal energetics and ATP depletion in pressure-overload mouse hearts: in vivo high-energy phosphate concentration measures by noninvasive magnetic resonance
- (2009) Ashish Gupta et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Cardiac phenotype of mitochondrial creatine kinase knockout mice is modified on a pure C57BL/6 genetic background
- (2008) Craig A. Lygate et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
Create your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create NowBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started