Prenatal exposure to cannabinoids evokes long-lasting functional alterations by targeting CB1receptors on developing cortical neurons

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Prenatal marijuana exposure predicts marijuana use in young adulthood

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Beliefs modulate the effects of drugs on the human brain: Fig. 1.

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Parental THC Exposure Leads to Compulsive Heroin-Seeking and Altered Striatal Synaptic Plasticity in the Subsequent Generation

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Endocannabinoids modulate cortical development by configuring Slit2/Robo1 signalling

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Cannabinoid CB1 Receptor in Dorsal Telencephalic Glutamatergic Neurons: Distinctive Sufficiency for Hippocampus-Dependent and Amygdala-Dependent Synaptic and Behavioral Functions

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The CB1 Cannabinoid Receptor Drives Corticospinal Motor Neuron Differentiation through the Ctip2/Satb2 Transcriptional Regulation Axis

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Endocannabinoids via CB1 receptors act as neurogenic niche cues during cortical development

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Requirement of cannabinoid CB1 receptors in cortical pyramidal neurons for appropriate development of corticothalamic and thalamocortical projections

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A cannabinoid analogue of Δ9-tetrahydrocannabinol disrupts neural development in chick

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Endocannabinoid signaling controls pyramidal cell specification and long-range axon patterning

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