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Title
Oxidative metabolism in YAC128 mouse model of Huntington's disease
Authors
Keywords
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Journal
HUMAN MOLECULAR GENETICS
Volume 24, Issue 17, Pages 4862-4878
Publisher
Oxford University Press (OUP)
Online
2015-06-04
DOI
10.1093/hmg/ddv209
References
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Related references
Note: Only part of the references are listed.- Huntingtin protein is essential for mitochondrial metabolism, bioenergetics and structure in murine embryonic stem cells
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- A role of mitochondrial complex II defects in genetic models of Huntington's disease expressing N-terminal fragments of mutant huntingtin
- (2013) Maria Damiano et al. HUMAN MOLECULAR GENETICS
- Choosing an animal model for the study of Huntington's disease
- (2013) Mahmoud A. Pouladi et al. NATURE REVIEWS NEUROSCIENCE
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- (2013) Caroline Gouarné et al. PLoS One
- Mitochondrial DNA damage Is associated with reduced mitochondrial bioenergetics in Huntington's disease
- (2012) Almas Siddiqui et al. FREE RADICAL BIOLOGY AND MEDICINE
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- (2012) A. Valencia et al. HUMAN MOLECULAR GENETICS
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- Abnormal mitochondrial dynamics, mitochondrial loss and mutant huntingtin oligomers in Huntington's disease: implications for selective neuronal damage
- (2011) U. Shirendeb et al. HUMAN MOLECULAR GENETICS
- Mutant huntingtin's interaction with mitochondrial protein Drp1 impairs mitochondrial biogenesis and causes defective axonal transport and synaptic degeneration in Huntington's disease
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- Mutant huntingtin binds the mitochondrial fission GTPase dynamin-related protein-1 and increases its enzymatic activity
- (2011) Wenjun Song et al. NATURE MEDICINE
- BAX insertion, oligomerization, and outer membrane permeabilization in brain mitochondria: Role of permeability transition and SH-redox regulation
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- Supercomplexes of the mitochondrial electron transport chain decline in the aging rat heart
- (2009) Luis A. Gómez et al. ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS
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- N-Terminal Mutant Huntingtin Associates with Mitochondria and Impairs Mitochondrial Trafficking
- (2008) A. L. Orr et al. JOURNAL OF NEUROSCIENCE
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