Increased expression of TREM2 in peripheral cells from mild cognitive impairment patients who progress into Alzheimer's disease
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Title
Increased expression of TREM2 in peripheral cells from mild cognitive impairment patients who progress into Alzheimer's disease
Authors
Keywords
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Journal
EUROPEAN JOURNAL OF NEUROLOGY
Volume 25, Issue 6, Pages 805-810
Publisher
Wiley
Online
2018-01-30
DOI
10.1111/ene.13583
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Note: Only part of the references are listed.- Higher Peripheral TREM2 mRNA Levels Relate to Cognitive Deficits and Hippocampal Atrophy in Alzheimer’s Disease and Amnestic Mild Cognitive Impairment
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- DNA methylation changes at TREM2 intron 1 and TREM2 mRNA expression in patients with Alzheimer's disease
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- Adenosine A2A Receptor Expression in Peripheral Blood Mononuclear Cells of Patients with Mild Cognitive Impairment
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- Cerebrospinal fluid soluble TREM2 is higher in Alzheimer disease and associated with mutation status
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- sTREM2 cerebrospinal fluid levels are a potential biomarker for microglia activity in early‐stage Alzheimer's disease and associate with neuronal injury markers
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- Gene promoter methylation and expression of Pin1 differ between patients with frontotemporal dementia and Alzheimer's disease
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- TREM2 Lipid Sensing Sustains the Microglial Response in an Alzheimer’s Disease Model
- (2015) Yaming Wang et al. CELL
- Apolipoprotein E Is a Ligand for Triggering Receptor Expressed on Myeloid Cells 2 (TREM2)
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- Triggering Receptor Expressed on Myeloid Cells 2 (TREM2) Deficiency Attenuates Phagocytic Activities of Microglia and Exacerbates Ischemic Damage in Experimental Stroke
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- Peripheral Blood Mononuclear Cells as a Laboratory to Study Dementia in the Elderly
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- Sequential Proteolytic Processing of the Triggering Receptor Expressed on Myeloid Cells-2 (TREM2) Protein by Ectodomain Shedding and γ-Secretase-dependent Intramembranous Cleavage
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- Differences of peripheral inflammatory markers between mild cognitive impairment and Alzheimer's disease
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