FXYD5 Is an Essential Mediator of the Inflammatory Response during Lung Injury

Hypaphorine Attenuates Lipopolysaccharide-Induced Endothelial Inflammation via Regulation of TLR4 and PPAR-γ Dependent on PI3K/Akt/mTOR Signal Pathway

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The contributions of lung macrophage and monocyte heterogeneity to influenza pathogenesis

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FXYD5 Protein Has a Pro-inflammatory Role in Epithelial Cells

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TheO-glycosylated ectodomain of FXYD5 impairs adhesion by disrupting cell–cell trans-dimerization of Na,K-ATPase β1subunits

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Macrophage-epithelial paracrine crosstalk inhibits lung edema clearance during influenza infection

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FXYD1 negatively regulates Na+/K+-ATPase activity in lung alveolar epithelial cells

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Microbe-inducible trafficking pathways that control Toll-like receptor signaling

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Alveolar Type II Epithelial Cells Contribute to the Anti-Influenza A Virus Response in the Lung by Integrating Pathogen- and Microenvironment-Derived Signals

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Pulmonary epithelial barrier function: some new players and mechanisms

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The Eosinophil in Infection

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Eosinophils in mucosal immune responses

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Corrigendum: The kinase Jnk2 promotes stress-induced mitophagy by targeting the small mitochondrial form of the tumor suppressor ARF for degradation

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Decreased CXCL12 is associated with impaired alveolar epithelial cell migration and poor lung healing after lung resection

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A Cross-Disciplinary Perspective on the Innate Immune Responses to Bacterial Lipopolysaccharide

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Innate sense of purpose for IKKβ: Fig. 1.

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 -Catenin is an inhibitor of transcription

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Nonclassical Ly6C− Monocytes Drive the Development of Inflammatory Arthritis in Mice

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Eosinophils in the Lung – Modulating Apoptosis and Efferocytosis in Airway Inflammation

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Novel concepts of acute lung injury and alveolar-capillary barrier dysfunction

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Flow Cytometric Analysis of Macrophages and Dendritic Cell Subsets in the Mouse Lung

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Pathogenesis of influenza-induced acute respiratory distress syndrome

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Suppression of inflammation and acute lung injury by Miz1 via repression of C/EBP-δ

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Leading Neutrophils to the Alveoli

(2012) Xiao Su   AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE

Monocytes Control Second-Phase Neutrophil Emigration in Established Lipopolysaccharide-induced Murine Lung Injury

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Dysadherin expression promotes the motility and survival of human breast cancer cells by AKT activation

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Alveolar Epithelial Cells Are Critical in Protection of the Respiratory Tract by Secretion of Factors Able To Modulate the Activity of Pulmonary Macrophages and Directly Control Bacterial Growth

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The Role of Alveolar Epithelial Cells in Initiating and Shaping Pulmonary Immune Responses: Communication between Innate and Adaptive Immune Systems

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Altered lung function relates to inflammation in an acute LPS mouse model

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TNF and ubiquitin at the crossroads of gene activation, cell death, inflammation, and cancer

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Lung-specific loss of the laminin α3 subunit confers resistance to mechanical injury

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Innate Immune Responses to Bacterial Ligands in the Peripheral Human Lung – Role of Alveolar Epithelial TLR Expression and Signalling

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Toll-Like Receptors: New Players in Myocardial Ischemia/Reperfusion Injury

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Respiratory epithelial cells in innate immunity to influenza virus infection

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Dysadherin can enhance tumorigenesis by conferring properties of stem-like cells to hepatocellular carcinoma cells

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FXYD5 modulates Na+ absorption and is increased in cystic fibrosis airway epithelia

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LPS/TLR4 signal transduction pathway

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Lipopolysaccharide sensing an important factor in the innate immune response to Gram-negative bacterial infections: Benefits and hazards of LPS hypersensitivity

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Significant Involvement of CCL2 (MCP-1) in Inflammatory Disorders of the Lung

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