Differentiation and Transmigration of CD4 T Cells in Neuroinflammation and Autoimmunity
Published 2017 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Differentiation and Transmigration of CD4 T Cells in Neuroinflammation and Autoimmunity
Authors
Keywords
-
Journal
Frontiers in Immunology
Volume 8, Issue -, Pages -
Publisher
Frontiers Media SA
Online
2017-11-29
DOI
10.3389/fimmu.2017.01695
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- IFN-γ promotes transendothelial migration of CD4+ T cells across the blood-brain barrier
- (2017) Sandip Ashok Sonar et al. IMMUNOLOGY AND CELL BIOLOGY
- Transendothelial migration: unifying principles from the endothelial perspective
- (2016) William A. Muller IMMUNOLOGICAL REVIEWS
- Interactions between genetic, lifestyle and environmental risk factors for multiple sclerosis
- (2016) Tomas Olsson et al. Nature Reviews Neurology
- Experimental priming of encephalitogenic Th1/Th17 cells requires pertussis toxin-driven IL-1β production by myeloid cells
- (2016) Francesca Ronchi et al. Nature Communications
- Inflammatory Th17 Cells Express Integrin α v β 3 for Pathogenic Function
- (2016) Fang Du et al. Cell Reports
- Internalization and presentation of myelin antigens by the brain endothelium guides antigen-specific T cell migration
- (2016) Melissa A Lopes Pinheiro et al. eLife
- Melanoma cell adhesion molecule-positive CD8 T lymphocytes mediate central nervous system inflammation
- (2015) Catherine Larochelle et al. ANNALS OF NEUROLOGY
- Cell surface levels of endothelial ICAM-1 influence the transcellular or paracellular T-cell diapedesis across the blood-brain barrier
- (2015) Michael Abadier et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- IL-12-polarized Th1 cells produce GM-CSF and induce EAE independent of IL-23
- (2015) Heather M. Grifka-Walk et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- Integration of Th17- and Lymphotoxin-Derived Signals Initiates Meningeal-Resident Stromal Cell Remodeling to Propagate Neuroinflammation
- (2015) Natalia B. Pikor et al. IMMUNITY
- Th17 cells transdifferentiate into regulatory T cells during resolution of inflammation
- (2015) Nicola Gagliani et al. NATURE
- Th17 cells give rise to Th1 cells that are required for the pathogenesis of colitis
- (2015) Stacey N. Harbour et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Revisiting the Mechanisms of CNS Immune Privilege
- (2015) Antoine Louveau et al. TRENDS IN IMMUNOLOGY
- CCR2 defines in vivo development and homing of IL-23-driven GM-CSF-producing Th17 cells
- (2015) Ervin E. Kara et al. Nature Communications
- Focal MMP-2 and MMP-9 Activity at the Blood-Brain Barrier Promotes Chemokine-Induced Leukocyte Migration
- (2015) Jian Song et al. Cell Reports
- Role of tumor necrosis factor superfamily in neuroinflammation and autoimmunity
- (2015) Sandip Sonar Frontiers in Immunology
- PSGL-1 and E/P-selectins are essential for T-cell rolling in inflamed CNS microvessels but dispensable for initiation of EAE
- (2014) Karthik Sathiyanadan et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- The Transcription Factors T-bet and Runx Are Required for the Ontogeny of Pathogenic Interferon-γ-Producing T Helper 17 Cells
- (2014) Yan Wang et al. IMMUNITY
- Once a Treg, always a Treg?
- (2014) Deepali V. Sawant et al. IMMUNOLOGICAL REVIEWS
- Cutting Edge: IL-21 Is Not Essential for Th17 Differentiation or Experimental Autoimmune Encephalomyelitis
- (2014) J. M. Coquet et al. JOURNAL OF IMMUNOLOGY
- Rapid Remodeling of Tight Junctions during Paracellular Diapedesis in a Human Model of the Blood–Brain Barrier
- (2014) Ryan C. Winger et al. JOURNAL OF IMMUNOLOGY
- Modeling the heterogeneity of multiple sclerosis in animals
- (2013) Sarah B. Simmons et al. TRENDS IN IMMUNOLOGY
- Role of miRNAs in CD4 T cell plasticity during inflammation and tolerance
- (2013) Apoorva Sethi et al. Frontiers in Genetics
- Melanoma cell adhesion molecule identifies encephalitogenic T lymphocytes and promotes their recruitment to the central nervous system
- (2012) Catherine Larochelle et al. BRAIN
- Induction and molecular signature of pathogenic TH17 cells
- (2012) Youjin Lee et al. NATURE IMMUNOLOGY
- Molecular Mechanisms of Treg-Mediated T Cell Suppression
- (2012) Angelika Schmidt et al. Frontiers in Immunology
- CXCR7 influences leukocyte entry into the CNS parenchyma by controlling abluminal CXCL12 abundance during autoimmunity
- (2011) Lillian Cruz-Orengo et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Th17 lymphocytes traffic to the central nervous system independently of α4 integrin expression during EAE
- (2011) Veit Rothhammer et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Cutting Edge: Loss of 4 Integrin Expression Differentially Affects the Homing of Th1 and Th17 Cells
- (2011) S. Glatigny et al. JOURNAL OF IMMUNOLOGY
- RORγt drives production of the cytokine GM-CSF in helper T cells, which is essential for the effector phase of autoimmune neuroinflammation
- (2011) Laura Codarri et al. NATURE IMMUNOLOGY
- The Hedgehog Pathway Promotes Blood-Brain Barrier Integrity and CNS Immune Quiescence
- (2011) J. I. Alvarez et al. SCIENCE
- The blood–brain barrier, chemokines and multiple sclerosis
- (2010) David W. Holman et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE
- Differential Roles for Endothelial ICAM-1, ICAM-2, and VCAM-1 in Shear-Resistant T Cell Arrest, Polarization, and Directed Crawling on Blood-Brain Barrier Endothelium
- (2010) O. Steiner et al. JOURNAL OF IMMUNOLOGY
- Generation of pathogenic TH17 cells in the absence of TGF-β signalling
- (2010) Kamran Ghoreschi et al. NATURE
- Effector T cell plasticity: flexibility in the face of changing circumstances
- (2010) Kenneth M Murphy et al. NATURE IMMUNOLOGY
- T-bet represses TH17 differentiation by preventing Runx1-mediated activation of the gene encoding RORγt
- (2010) Vanja Lazarevic et al. NATURE IMMUNOLOGY
- Epigenetic mechanisms of regulation of Foxp3 expression
- (2009) G. Lal et al. BLOOD
- Antigen-Specific Dependence of Tr1-Cell Therapy in Preclinical Models of Islet Transplant
- (2009) N. Gagliani et al. DIABETES
- T-bet is essential for encephalitogenicity of both Th1 and Th17 cells
- (2009) Yuhong Yang et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Transcellular migration of leukocytes is mediated by the endothelial lateral border recycling compartment
- (2009) Zahra Mamdouh et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Th1, Th17, and Th9 Effector Cells Induce Experimental Autoimmune Encephalomyelitis with Different Pathological Phenotypes
- (2009) A. Jager et al. JOURNAL OF IMMUNOLOGY
- Cutting Edge: IL-23 Receptor GFP Reporter Mice Reveal Distinct Populations of IL-17-Producing Cells
- (2009) Amit Awasthi et al. JOURNAL OF IMMUNOLOGY
- Endothelial basement membrane laminin α5 selectively inhibits T lymphocyte extravasation into the brain
- (2009) Chuan Wu et al. NATURE MEDICINE
- IL‐21 and IL‐21R are not required for development of Th17 cells and autoimmunityin vivo
- (2008) Ivo Sonderegger et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- Signal Transducer and Activator of Transcription 4 Is Required for the Transcription Factor T-bet to Promote T Helper 1 Cell-Fate Determination
- (2008) Vivian T. Thieu et al. IMMUNITY
- IL-17A and IL-17F do not contribute vitally to autoimmune neuro-inflammation in mice
- (2008) Stefan Haak et al. JOURNAL OF CLINICAL INVESTIGATION
- ICAM-1–mediated Endothelial Nitric Oxide Synthase Activation via Calcium and AMP-activated Protein Kinase Is Required for Transendothelial Lymphocyte Migration
- (2008) Roberta Martinelli et al. MOLECULAR BIOLOGY OF THE CELL
- TGF-β-induced Foxp3 inhibits TH17 cell differentiation by antagonizing RORγt function
- (2008) Liang Zhou et al. NATURE
- Natural and TGF-β–induced Foxp3+CD4+ CD25+ regulatory T cells are not mirror images of each other
- (2008) David A. Horwitz et al. TRENDS IN IMMUNOLOGY
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExplorePublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More