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Title
Induction and molecular signature of pathogenic TH17 cells
Authors
Keywords
-
Journal
NATURE IMMUNOLOGY
Volume 13, Issue 10, Pages 991-999
Publisher
Springer Nature
Online
2012-09-10
DOI
10.1038/ni.2416
References
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Related references
Note: Only part of the references are listed.- Cutting Edge: Th1 Cells Facilitate the Entry of Th17 Cells to the Central Nervous System during Experimental Autoimmune Encephalomyelitis
- (2014) R. A. O'Connor et al. JOURNAL OF IMMUNOLOGY
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- The encephalitogenicity of TH17 cells is dependent on IL-1- and IL-23-induced production of the cytokine GM-CSF
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- IL-7Rα confers susceptibility to experimental autoimmune encephalomyelitis
- (2010) C C Walline et al. GENES AND IMMUNITY
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- (2010) Kamran Ghoreschi et al. NATURE
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- Crucial role of interleukin-7 in T helper type 17 survival and expansion in autoimmune disease
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- IL-17 and Th17 Cells
- (2009) Thomas Korn et al. Annual Review of Immunology
- T-bet is essential for encephalitogenicity of both Th1 and Th17 cells
- (2009) Yuhong Yang et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Th1, Th17, and Th9 Effector Cells Induce Experimental Autoimmune Encephalomyelitis with Different Pathological Phenotypes
- (2009) A. Jager et al. JOURNAL OF IMMUNOLOGY
- Cutting Edge: IL-23 Receptor GFP Reporter Mice Reveal Distinct Populations of IL-17-Producing Cells
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- The interleukin 23 receptor is essential for the terminal differentiation of interleukin 17–producing effector T helper cells in vivo
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- Association of interleukin-23 receptor variants with ankylosing spondylitis
- (2008) Proton Rahman et al. ARTHRITIS AND RHEUMATISM
- Tissue-specific expression of Cre recombinase from theTgfb3 locus
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- The aryl hydrocarbon receptor links TH17-cell-mediated autoimmunity to environmental toxins
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