T-bet is essential for encephalitogenicity of both Th1 and Th17 cells

Host T Cells Are the Main Producers of IL-17 within the Central Nervous System during Initiation of Experimental Autoimmune Encephalomyelitis Induced by Adoptive Transfer of Th1 Cell Lines

(2014) J. R. Lees et al.   JOURNAL OF IMMUNOLOGY

Overlapping and Distinct Roles of STAT4 and T-bet in the Regulation of T Cell Differentiation and Allergic Airway Inflammation

(2014) S. Furuta et al.   JOURNAL OF IMMUNOLOGY

Late Developmental Plasticity in the T Helper 17 Lineage

(2009) Yun Kyung Lee et al.   IMMUNITY

IL-17A and IL-17F do not contribute vitally to autoimmune neuro-inflammation in mice

(2008) Stefan Haak et al.   JOURNAL OF CLINICAL INVESTIGATION

T-bet Deficiency Attenuates Renal Injury in Experimental Crescentic Glomerulonephritis

(2008) R. K. S. Phoon et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

Differential regulation of central nervous system autoimmunity by TH1 and TH17 cells

(2008) Ingunn M Stromnes et al.   NATURE MEDICINE