A novel Dock8 gene mutation confers diabetogenic susceptibility in the LEW.1AR1/Ztm-iddm rat, an animal model of human type 1 diabetes
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Title
A novel Dock8 gene mutation confers diabetogenic susceptibility in the LEW.1AR1/Ztm-iddm rat, an animal model of human type 1 diabetes
Authors
Keywords
Animal model, Mutation, T cells, Type 1 diabetes
Journal
DIABETOLOGIA
Volume 58, Issue 12, Pages 2800-2809
Publisher
Springer Nature
Online
2015-09-12
DOI
10.1007/s00125-015-3757-7
References
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Related references
Note: Only part of the references are listed.- Dock-family exchange factors in cell migration and disease
- (2014) Gilles Gadea et al. EUROPEAN JOURNAL OF CELL BIOLOGY
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- (2013) G. Crawford et al. BLOOD
- Islet infiltration, cytokine expression and beta cell death in the NOD mouse, BB rat, Komeda rat, LEW.1AR1-iddm rat and humans with type 1 diabetes
- (2013) Anne Jörns et al. DIABETOLOGIA
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- (2013) Melissa C. Mizesko et al. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
- Dedicator of Cytokinesis 8 Interacts with Talin and Wiskott-Aldrich Syndrome Protein To Regulate NK Cell Cytotoxicity
- (2013) H. Ham et al. JOURNAL OF IMMUNOLOGY
- DOCK8 is a Cdc42 activator critical for interstitial dendritic cell migration during immune responses
- (2012) Y. Harada et al. BLOOD
- The Mst1 and Mst2 kinases control activation of rho family GTPases and thymic egress of mature thymocytes
- (2012) Fan Mou et al. JOURNAL OF EXPERIMENTAL MEDICINE
- DOCK8 functions as an adaptor that links TLR-MyD88 signaling to B cell activation
- (2012) Haifa H Jabara et al. NATURE IMMUNOLOGY
- DOCK8 is essential for T-cell survival and the maintenance of CD8+ T-cell memory
- (2011) Teresa Lambe et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- Dedicator of cytokinesis 8 (DOCK8) deficiency
- (2010) Helen C Su Current Opinion in Allergy and Clinical Immunology
- Diabetes Prevention by Immunomodulatory FTY720 Treatment in the LEW.1AR1-iddmRat Despite Immune Cell Activation
- (2010) Anne Jörns et al. ENDOCRINOLOGY
- Prevention of spontaneous immune-mediated diabetes development in the LEW.1AR1-iddm rat by selective CD8+ T cell transfer is associated with a cytokine shift in the pancreas-draining lymph nodes
- (2009) T. Arndt et al. DIABETOLOGIA
- Dock8 mutations cripple B cell immunological synapses, germinal centers and long-lived antibody production
- (2009) Katrina L Randall et al. NATURE IMMUNOLOGY
- Combined Immunodeficiency Associated withDOCK8Mutations
- (2009) Qian Zhang et al. NEW ENGLAND JOURNAL OF MEDICINE
- Structural specializations of A2, a force-sensing domain in the ultralarge vascular protein von Willebrand factor
- (2009) Q. Zhang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Activation of Rho GTPases by DOCK Exchange Factors Is Mediated by a Nucleotide Sensor
- (2009) J. Yang et al. SCIENCE
- Impaired survival of peripheral T cells, disrupted NK/NKT cell development, and liver failure in mice lacking Gimap5
- (2008) R. D. Schulteis et al. BLOOD
- A genome-wide SNP panel for mapping and association studies in the rat
- (2008) Isaäc J Nijman et al. BMC GENOMICS
- Cellular signaling for activation of Rho GTPase Cdc42
- (2008) Soniya Sinha et al. CELLULAR SIGNALLING
- The mutation of the LEW.1AR1-iddm rat maps to the telomeric end of rat chromosome 1
- (2008) Heike Weiss et al. MAMMALIAN GENOME
- Dock10, a novel CZH protein selectively induced by interleukin-4 in human B lymphocytes
- (2008) Estefanía Yelo et al. MOLECULAR IMMUNOLOGY
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