DOCK8 functions as an adaptor that links TLR-MyD88 signaling to B cell activation
Published 2012 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
DOCK8 functions as an adaptor that links TLR-MyD88 signaling to B cell activation
Authors
Keywords
-
Journal
NATURE IMMUNOLOGY
Volume 13, Issue 6, Pages 612-620
Publisher
Springer Nature America, Inc
Online
2012-05-14
DOI
10.1038/ni.2305
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- MyD88 Signaling Is Not Essential for Induction of Antigen-Specific B Cell Responses but Is Indispensable for Protection against Streptococcus pneumoniae Infection following Oral Vaccination with Attenuated Salmonella Expressing PspA Antigen
- (2014) S.-M. Park et al. JOURNAL OF IMMUNOLOGY
- STAT3-Mediated Up-Regulation of BLIMP1 Is Coordinated with BCL6 Down-Regulation to Control Human Plasma Cell Differentiation
- (2014) S. A. Diehl et al. JOURNAL OF IMMUNOLOGY
- Selective Utilization of Toll-like Receptor and MyD88 Signaling in B Cells for Enhancement of the Antiviral Germinal Center Response
- (2011) Baidong Hou et al. IMMUNITY
- Toll-like receptor driven B cell activation in the induction of systemic autoimmunity
- (2011) Nathaniel M. Green et al. SEMINARS IN IMMUNOLOGY
- MyD88 signal is required for more efficient induction of Ag-specific adaptive immune responses and antitumor resistance in a human papillomavirus E7 DNA vaccine model
- (2011) Jeong-Im Sin VACCINE
- Memory B and memory plasma cells
- (2010) Taketoshi Yoshida et al. IMMUNOLOGICAL REVIEWS
- B cell–intrinsic signaling through IL-21 receptor and STAT3 is required for establishing long-lived antibody responses in humans
- (2010) Danielle T. Avery et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Selective control of type I IFN induction by the Rac activator DOCK2 during TLR-mediated plasmacytoid dendritic cell activation
- (2010) Kazuhito Gotoh et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Phosphoproteomic characterization of PYK2 signaling pathways involved in osteogenesis
- (2010) Peter C. Bonnette et al. Journal of Proteomics
- STAT3 is a substrate of SYK tyrosine kinase in B-lineage leukemia/lymphoma cells exposed to oxidative stress
- (2010) F. M. Uckun et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- TLR2-, TLR4- and Myd88-independent acquired humoral and cellular immunity against Salmonella enterica serovar Typhimurium
- (2009) Sabine A. Seibert et al. IMMUNOLOGY LETTERS
- Large deletions and point mutations involving the dedicator of cytokinesis 8 (DOCK8) in the autosomal-recessive form of hyper-IgE syndrome
- (2009) Karin R. Engelhardt et al. JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY
- IFN-α amplifies human naïve B cell TLR-9-mediated activation and Ig production
- (2009) L. Giordani et al. JOURNAL OF LEUKOCYTE BIOLOGY
- PYK2 interacts with MyD88 and regulates MyD88-mediated NF- B activation in macrophages
- (2009) C.-X. Xi et al. JOURNAL OF LEUKOCYTE BIOLOGY
- Activation of human B cells by the agonist CD40 antibody CP-870,893 and augmentation with simultaneous toll-like receptor 9 stimulation
- (2009) Erica L Carpenter et al. Journal of Translational Medicine
- Dock8 mutations cripple B cell immunological synapses, germinal centers and long-lived antibody production
- (2009) Katrina L Randall et al. NATURE IMMUNOLOGY
- Combined Immunodeficiency Associated withDOCK8Mutations
- (2009) Qian Zhang et al. NEW ENGLAND JOURNAL OF MEDICINE
- Myd88 Is Required for an Antibody Response to Retroviral Infection
- (2009) Edward P. Browne et al. PLoS Pathogens
- Expression of Angiotensin-converting Enzyme Changes Major Histocompatibility Complex Class I Peptide Presentation by Modifying C Termini of Peptide Precursors
- (2008) Xiao Z. Shen et al. JOURNAL OF BIOLOGICAL CHEMISTRY
Find the ideal target journal for your manuscript
Explore over 38,000 international journals covering a vast array of academic fields.
SearchAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started