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The antineoplastic drug flavopiridol reverses memory impairment induced by Amyloid-ß 1-42 oligomers in mice

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Hippocampal Injections of Oligomeric Amyloid β-peptide (1–42) Induce Selective Working Memory Deficits and Long-lasting Alterations of ERK Signaling Pathway

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Acute amnestic encephalopathy in amyloid-β oligomer–injected mice is due to their widespread diffusion in vivo

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Systematic analysis of time-dependent neural effects of soluble amyloid β oligomers in culture and in vivo: Prevention by scyllo-inositol

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Methanolic Extract of Piper nigrum Fruits Improves Memory Impairment by Decreasing Brain Oxidative Stress in Amyloid Beta(1–42) Rat Model of Alzheimer’s Disease

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Alzheimer's Disease-Like Pathology Induced by Amyloid-β Oligomers in Nonhuman Primates

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Comparative efficacy of Ocimum sanctum L. and Ocimum basilicum L. essential oils against amyloid beta (1–42)-induced anxiety and depression in laboratory rats

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Granulocyte-Colony Stimulating Factor Attenuates Oligomeric Amyloid β Neurotoxicity by Activation of Neprilysin

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Memantine Rescues Transient Cognitive Impairment Caused by High-Molecular-Weight A  Oligomers But Not the Persistent Impairment Induced by Low-Molecular-Weight Oligomers

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7,8-Dihydroxyflavone Prevents Synaptic Loss and Memory Deficits in a Mouse Model of Alzheimer’s Disease

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Alzheimer's Disease Related Markers, Cellular Toxicity and Behavioral Deficits Induced Six Weeks after Oligomeric Amyloid-β Peptide Injection in Rats

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Amyloid β-Peptide (1–42)-Induced Oxidative Stress in Alzheimer Disease: Importance in Disease Pathogenesis and Progression

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Effects of Perceived Long-Term Stress on Subjective and Objective Aspects of Memory and Cognitive Functioning in a Middle-Aged Population-Based Sample

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Neurotoxicity and Memory Deficits Induced by Soluble Low-Molecular-Weight Amyloid- 1-42 Oligomers Are Revealed In Vivo by Using a Novel Animal Model

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Soluble A  Promotes Wild-Type Tau Pathology In Vivo

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Cognitive dysfunction in psychiatric disorders: characteristics, causes and the quest for improved therapy

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Deregulation of hypothalamic-pituitary-adrenal axis functions in an Alzheimer's disease rat model

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Time-Course and Regional Analyses of the Physiopathological Changes Induced after Cerebral Injection of an Amyloid β Fragment in Rats

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Moderate Grade Hyperammonemia Induced Concordant Activation of Antioxidant Enzymes is Associated with Prevention of Oxidative Stress in the Brain Slices

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Phenylbutyrate rescues dendritic spine loss associated with memory deficits in a mouse model of Alzheimer disease

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A causative link between the structure of aberrant protein oligomers and their toxicity

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Amyloid-β–induced neuronal dysfunction in Alzheimer's disease: from synapses toward neural networks

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Preparation and characterization of toxic Aβ aggregates for structural and functional studies in Alzheimer's disease research

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Amyloid β-peptide levels in laser capture microdissected cornu ammonis 1 pyramidal neurons of Alzheimerʼs brain

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Synthetic amyloid-  oligomers impair long-term memory independently of cellular prion protein

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Intracellular accumulation of amyloid-beta – a predictor for synaptic dysfunction and neuron loss in Alzheimer’s disease

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Oxidative Stress and Neurodegenerative Diseases: A Review of Upstream and Downstream Antioxidant Therapeutic Options

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Depression and Alzheimer's disease: Neurobiological links and common pharmacological targets

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Comparison of Alzheimer A (1-40) and A (1-42) amyloid fibrils reveals similar protofilament structures

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Soluble oligomers of the amyloid β-protein impair synaptic plasticity and behavior

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Epigenetic targets of HDAC inhibition in neurodegenerative and psychiatric disorders

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Amyloid-cholinesterase interactions

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The neuroprotective role of melatonin against amyloidβpeptide injected mice

(2008) J. Gunasingh Masilamoni et al.   FREE RADICAL RESEARCH

Parenchymal and vascular Aβ-deposition and its effects on the degeneration of neurons and cognition in Alzheimer's disease

(2008) Dietmar Rudolf Thal et al.   JOURNAL OF CELLULAR AND MOLECULAR MEDICINE

Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

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