Dendritic cells resist to disulfiram-induced cytotoxicity, but reduced interleukin-12/23(p40) production

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Targeting FROUNT with disulfiram suppresses macrophage accumulation and its tumor-promoting properties

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JAK inhibitors impair GM-CSF-mediated signaling in innate immune cells

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Disulfiram’s anti-cancer activity reflects targeting NPL4, not inhibition of aldehyde dehydrogenase

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Disulfiram targeting lymphoid malignant cell lines via ROS-JNK activation as well as Nrf2 and NF-kB pathway inhibition

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Disulfiram modulated ROS–MAPK and NFκB pathways and targeted breast cancer cells with cancer stem cell-like properties

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