CD36 regulates LPS-induced acute lung injury by promoting macrophages M1 polarization
Published 2022 View Full Article
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Title
CD36 regulates LPS-induced acute lung injury by promoting macrophages M1 polarization
Authors
Keywords
CD36, Macrophage polarization, Acute lung injury, NF-κB
Journal
CELLULAR IMMUNOLOGY
Volume 372, Issue -, Pages 104475
Publisher
Elsevier BV
Online
2022-01-12
DOI
10.1016/j.cellimm.2021.104475
References
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- (2020) Anna Ciesielska et al. CELLULAR AND MOLECULAR LIFE SCIENCES
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- (2016) Magdalena Hryhorowicz et al. ARCHIVUM IMMUNOLOGIAE ET THERAPIAE EXPERIMENTALIS
- Lipid rafts in immune signalling: current progress and future perspective
- (2016) Pallavi Varshney et al. IMMUNOLOGY
- Synthetic Amphipathic Helical Peptides Targeting CD36 Attenuate Lipopolysaccharide-Induced Inflammation and Acute Lung Injury
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- (2016) Tolani F. Olonisakin et al. JOURNAL OF INFECTIOUS DISEASES
- Diverse macrophage populations mediate acute lung inflammation and resolution
- (2014) Neil R. Aggarwal et al. AMERICAN JOURNAL OF PHYSIOLOGY-LUNG CELLULAR AND MOLECULAR PHYSIOLOGY
- Co-operation of TLR4 and raft proteins in LPS-induced pro-inflammatory signaling
- (2014) Agnieszka Płóciennikowska et al. CELLULAR AND MOLECULAR LIFE SCIENCES
- oxLDL induces inflammatory responses in vascular smooth muscle cells via urokinase receptor association with CD36 and TLR4
- (2013) Yulia Kiyan et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Pulmonary Macrophage Subpopulations in the Induction and Resolution of Acute Lung Injury
- (2012) Laura K. Johnston et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
- CD36 level and trafficking are determinants of lipolysis in adipocytes
- (2012) Dequan Zhou et al. FASEB JOURNAL
- LPS-induced chemokine expression in both MyD88-dependent and -independent manners is regulated by Cot/Tpl2-ERK axis in macrophages
- (2012) Kenjiro Bandow et al. FEBS LETTERS
- Propofol attenuates lipopolysaccharide-induced monocyte chemoattractant protein-1 production through p38 MAPK and SAPK/JNK in alveolar epithelial cells
- (2012) Liguo Wei et al. Journal of Anesthesia
- Distinct Macrophage Subpopulations Characterize Acute Infection and Chronic Inflammatory Lung Disease
- (2012) M. Duan et al. JOURNAL OF IMMUNOLOGY
- Alternative activation of macrophages by IL-4 requires SHIP degradation
- (2011) Shelley B. Weisser et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- Cot/tpl2 activity is required for TLR-induced activation of the Akt p70 S6k pathway in macrophages: Implications for NO synthase 2 expression
- (2011) Marta López-Peláez et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- Class B Scavenger Receptor Types I and II and CD36 Mediate Bacterial Recognition and Proinflammatory Signaling Induced by Escherichia coli, Lipopolysaccharide, and Cytosolic Chaperonin 60
- (2011) I. N. Baranova et al. JOURNAL OF IMMUNOLOGY
- Unknown
- (2011) Jochen Grommes et al. MOLECULAR MEDICINE
- The value of the lipopolysaccharide-induced acute lung injury model in respiratory medicine
- (2010) Hong Chen et al. Expert Review of Respiratory Medicine
- Macrophages and fibroblasts during inflammation, tissue damage and organ injury
- (2009) Trevor Glaros Frontiers in Bioscience-Landmark
- CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer
- (2009) Cameron R Stewart et al. NATURE IMMUNOLOGY
- CD36, a Scavenger Receptor Involved in Immunity, Metabolism, Angiogenesis, and Behavior
- (2009) R. L. Silverstein et al. Science Signaling
- Cellular fatty acid uptake: a pathway under construction
- (2009) Xiong Su et al. TRENDS IN ENDOCRINOLOGY AND METABOLISM
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- (2008) Yong-Chen Lu et al. CYTOKINE
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- (2007) Fernando Oneissi Martinez Frontiers in Bioscience-Landmark
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