Mitochondrial abnormalities: a hub in metabolic syndrome-related cardiac dysfunction caused by oxidative stress
Published 2021 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Mitochondrial abnormalities: a hub in metabolic syndrome-related cardiac dysfunction caused by oxidative stress
Authors
Keywords
-
Journal
HEART FAILURE REVIEWS
Volume -, Issue -, Pages -
Publisher
Springer Science and Business Media LLC
Online
2021-05-05
DOI
10.1007/s10741-021-10109-6
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Mitochondrial and Nuclear DNA Oxidative Damage in Physiological and Pathological Aging
- (2020) Marta Kowalska et al. DNA AND CELL BIOLOGY
- Mesalazine Induces Oxidative Stress and Cytochrome c Release in Isolated Rat Heart Mitochondria: An Analysis of Cardiotoxic Effects
- (2020) Ahmad Salimi et al. INTERNATIONAL JOURNAL OF TOXICOLOGY
- Structure and mechanism of the mitochondrial Ca2+ uniporter holocomplex
- (2020) Minrui Fan et al. NATURE
- Qiliqiangxin Attenuates Oxidative Stress-Induced Mitochondrion-Dependent Apoptosis in Cardiomyocytes via PI3K/AKT/GSK3β Signaling Pathway
- (2019) Qifei Zhao et al. BIOLOGICAL & PHARMACEUTICAL BULLETIN
- Diverse functions of DNA glycosylases processing oxidative base lesions in brain
- (2019) Katja Scheffler et al. DNA REPAIR
- The Role of Signaling Pathways of Inflammation and Oxidative Stress in Development of Senescence and Aging Phenotypes in Cardiovascular Disease
- (2019) John Papaconstantinou Cells
- Leonurine protects cardiac function following acute myocardial infarction through anti‑apoptosis by the PI3K/AKT/GSK3β signaling pathway
- (2018) Lin Xu et al. Molecular Medicine Reports
- Cytoprotective mechanisms of DJ-1 against oxidative stress through modulating ERK1/2 and ASK1 signal transduction
- (2018) Stephanie E. Oh et al. Redox Biology
- PI3K–AKT Pathway Protects Cardiomyocytes Against Hypoxia-Induced Apoptosis by MitoKATP-Mediated Mitochondrial Translocation of pAKT
- (2018) Hua-Pei Song et al. CELLULAR PHYSIOLOGY AND BIOCHEMISTRY
- Therapeutic Effects of Coccomyxagloeobotrydiformis on the Metabolic Syndrome in Rats
- (2018) Ningning Zheng et al. CELLULAR PHYSIOLOGY AND BIOCHEMISTRY
- Targeting mitochondrial dysfunction and oxidative stress in heart failure: Challenges and opportunities
- (2018) Ligia Akemi Kiyuna et al. FREE RADICAL BIOLOGY AND MEDICINE
- Betulinic acid as apoptosis activator: Molecular mechanisms, mathematical modeling and chemical modifications
- (2018) Pranesh Kumar et al. LIFE SCIENCES
- α-Bisabolol abrogates isoproterenol-induced myocardial infarction by inhibiting mitochondrial dysfunction and intrinsic pathway of apoptosis in rats
- (2018) Mohamed Fizur Nagoor Meeran et al. MOLECULAR AND CELLULAR BIOCHEMISTRY
- Betulinic acid improves insulin sensitivity, hyperglycemia, inflammation and oxidative stress in metabolic syndrome rats via PI3K/Akt pathways
- (2018) R. A. Ajala-Lawal et al. ARCHIVES OF PHYSIOLOGY AND BIOCHEMISTRY
- Long-Term Regulation of Excitation–Contraction Coupling and Oxidative Stress in Cardiac Myocytes by Pirfenidone
- (2018) Adrián Monsalvo-Villegas et al. Frontiers in Physiology
- Role of carvacrol in cardioprotection against myocardial ischemia/reperfusion injury in rats through activation of MAPK/ERK and Akt/eNOS signaling pathways
- (2017) Yunping Chen et al. EUROPEAN JOURNAL OF PHARMACOLOGY
- Apoptosis in ischemic heart disease
- (2017) Elena Teringova et al. Journal of Translational Medicine
- Oxidative stress and mitochondrial dysfunction-linked neurodegenerative disorders
- (2016) Md. Torequl Islam NEUROLOGICAL RESEARCH
- ROS and ROS-Mediated Cellular Signaling
- (2016) Jixiang Zhang et al. Oxidative Medicine and Cellular Longevity
- Oxidative stress in cardiomyocytes contributes to decreased SERCA2a activity in rats with metabolic syndrome
- (2013) Jaime Balderas-Villalobos et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Activated apoptotic and anti-survival effects on rat hearts with fructose induced metabolic syndrome
- (2013) Shiu-Min Cheng et al. CELL BIOCHEMISTRY AND FUNCTION
- Cardiac Dysfunction and Oxidative Stress in the Metabolic Syndrome: an Update on Antioxidant Therapies
- (2013) Olesya Ilkun et al. CURRENT PHARMACEUTICAL DESIGN
- Role of oxidants on calcium and sodium movement in healthy and diseased cardiac myocytes
- (2013) Can M. Sag et al. FREE RADICAL BIOLOGY AND MEDICINE
- Redox modification of ryanodine receptors by mitochondria-derived reactive oxygen species contributes to aberrant Ca2+handling in ageing rabbit hearts
- (2013) Leroy L. Cooper et al. JOURNAL OF PHYSIOLOGY-LONDON
- Inhibition of JNK Mitochondrial Localization and Signaling Is Protective against Ischemia/Reperfusion Injury in Rats
- (2012) Jeremy W. Chambers et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Mitochondrial proteome remodelling in pressure overload-induced heart failure: the role of mitochondrial oxidative stress
- (2011) Dao-Fu Dai et al. CARDIOVASCULAR RESEARCH
- The multiple functions of cytochrome c and their regulation in life and death decisions of the mammalian cell: From respiration to apoptosis
- (2011) Maik Hüttemann et al. MITOCHONDRION
- Proteomic remodelling of mitochondrial oxidative pathways in pressure overload-induced heart failure
- (2009) Heiko Bugger et al. CARDIOVASCULAR RESEARCH
- Melatonin inhibits cardiolipin peroxidation in mitochondria and prevents the mitochondrial permeability transition and cytochrome c release
- (2009) Giuseppe Petrosillo et al. FREE RADICAL BIOLOGY AND MEDICINE
- Oxidative stress induces degradation of mitochondrial DNA
- (2009) Inna Shokolenko et al. NUCLEIC ACIDS RESEARCH
- Molecular mechanisms for myocardial mitochondrial dysfunction in the metabolic syndrome
- (2008) Heiko Bugger et al. CLINICAL SCIENCE
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started