LOXL2 Expression Status Is Correlated With Molecular Characterizations of Cervical Carcinoma and Associated With Poor Cancer Survival via Epithelial-Mesenchymal Transition (EMT) Phenotype

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MEXPRESS update 2019

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Perspective: APOBEC mutagenesis in drug resistance and immune escape in HIV and cancer evolution

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A Comprehensive Pan-Cancer Molecular Study of Gynecologic and Breast Cancers

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Association of Ipilimumab With Safety and Antitumor Activity in Women With Metastatic or Recurrent Human Papillomavirus–Related Cervical Carcinoma

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Safety and Efficacy of Pembrolizumab in Advanced, Programmed Death Ligand 1–Positive Cervical Cancer: Results From the Phase Ib KEYNOTE-028 Trial

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LOXL2 drives epithelial-mesenchymal transition via activation of IRE1-XBP1 signalling pathway

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Lysyl oxidase-like 2 represses Notch1 expression in the skin to promote squamous cell carcinoma progression

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Targeting copper to treat breast cancer

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Precise Classification of Cervical Carcinomas Combined with Somatic Mutation Profiling Contributes to Predicting Disease Outcome

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Lysyl oxidase-like 2 is critical to tumor microenvironment and metastatic niche formation in hepatocellular carcinoma

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Extracellular matrix density promotes EMT by weakening cell–cell adhesions

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LOXL2 expression is associated with invasiveness and negatively influences survival in breast cancer patients

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Lysyl oxidase-like-2 promotes tumour angiogenesis and is a potential therapeutic target in angiogenic tumours

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MCF-7 Cells Expressing Nuclear Associated Lysyl Oxidase-like 2 (LOXL2) Exhibit an Epithelial-to-Mesenchymal Transition (EMT) Phenotype and Are Highly Invasivein Vitro

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Lysyl oxidase-like 2 (LOXL2), a new regulator of cell polarity required for metastatic dissemination of basal-like breast carcinomas

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The Role of Lysyl Oxidase in SRC-Dependent Proliferation and Metastasis of Colorectal Cancer

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Secreted LOXL2 is a novel therapeutic target that promotes gastric cancer metastasis via the Src/FAK pathway

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Lysyl Oxidase-Like 2 as a New Poor Prognosis Marker of Squamous Cell Carcinomas

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New Roles for Copper Metabolism in Cell Proliferation, Signaling, and Disease

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