- Home
- Publications
- Publication Search
- Publication Details
Title
Role of NLRP3 inflammasome in inflammatory bowel diseases
Authors
Keywords
-
Journal
WORLD JOURNAL OF GASTROENTEROLOGY
Volume 25, Issue 33, Pages 4796-4804
Publisher
Baishideng Publishing Group Inc.
Online
2019-09-04
DOI
10.3748/wjg.v25.i33.4796
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Targeting NLRP3 Inflammasome in Inflammatory Bowel Disease: Putting out the Fire of Inflammation
- (2019) Bo-Zong Shao et al. INFLAMMATION
- Therapeutic potential of dinitrobenzene sulfonic acid (DNBS)-induced colitis in mice by targeting IL-1β and IL-18
- (2018) Daniela Impellizzeri et al. BIOCHEMICAL PHARMACOLOGY
- Loss-of-function CARD8 mutation causes NLRP3 inflammasome activation and Crohn’s disease
- (2018) Liming Mao et al. JOURNAL OF CLINICAL INVESTIGATION
- Updated review on immune factors in pathogenesis of Crohn’s disease
- (2018) Na Li et al. WORLD JOURNAL OF GASTROENTEROLOGY
- MCC950, a specific small molecule inhibitor of NLRP3 inflammasome attenuates colonic inflammation in spontaneous colitis mice
- (2018) Agampodi Promoda Perera et al. Scientific Reports
- Nucleotide-Binding Domain Leucine-Rich Repeat Containing Proteins and Intestinal Microbiota: Pivotal Players in Colitis and Colitis-Associated Cancer Development
- (2018) Anna Prossomariti et al. Frontiers in Immunology
- Curcumin as a therapeutic agent for blocking NF-κB activation in ulcerative colitis
- (2018) Yiqing Wang et al. IMMUNOPHARMACOLOGY AND IMMUNOTOXICOLOGY
- Use of Curcumin in Achieving Clinical and Endoscopic Remission in Ulcerative Colitis: A Systematic Review and Meta-analysis
- (2018) Umair Iqbal et al. AMERICAN JOURNAL OF THE MEDICAL SCIENCES
- NLRP3-Dependent and -Independent Processing of Interleukin (IL)-1β in Active Ulcerative Colitis
- (2018) Nicole Ranson et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
- The Role of NLRP3 and IL-1β in the Pathogenesis of Inflammatory Bowel Disease
- (2018) Liming Mao et al. Frontiers in Immunology
- Myeloid-derived miR-223 regulates intestinal inflammation via repression of the NLRP3 inflammasome
- (2017) Viola Neudecker et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Pyroptosis: Gasdermin-Mediated Programmed Necrotic Cell Death
- (2017) Jianjin Shi et al. TRENDS IN BIOCHEMICAL SCIENCES
- Remodelling of the gut microbiota by hyperactive NLRP3 induces regulatory T cells to maintain homeostasis
- (2017) Xiaomin Yao et al. Nature Communications
- Epithelial Cell Inflammasomes in Intestinal Immunity and Inflammation
- (2017) Andrea C. Lei-Leston et al. Frontiers in Immunology
- Cholecalciterol cholesterol emulsion ameliorates experimental colitis via down-regulating the pyroptosis signaling pathway
- (2016) Yangyang Xiong et al. EXPERIMENTAL AND MOLECULAR PATHOLOGY
- The Pathogenic Role of NLRP3 Inflammasome Activation in Inflammatory Bowel Diseases of Both Mice and Humans
- (2016) Ling Liu et al. Journal of Crohns & Colitis
- Association between functional NLRP3 polymorphisms and susceptibility to autoimmune and inflammatory diseases: a meta-analysis
- (2016) Y H Lee et al. LUPUS
- The NLRP3 inflammasome in kidney disease and autoimmunity
- (2016) Holly L Hutton et al. NEPHROLOGY
- NLRP3 inflammasome has a protective effect against oxazolone-induced colitis: a possible role in ulcerative colitis
- (2016) Shigehiro Itani et al. Scientific Reports
- The hierarchical structural architecture of inflammasomes, supramolecular inflammatory machines
- (2015) Arthur V Hauenstein et al. CURRENT OPINION IN STRUCTURAL BIOLOGY
- The interleukin (IL)-1 cytokine family – Balance between agonists and antagonists in inflammatory diseases
- (2015) Jennifer Palomo et al. CYTOKINE
- Assessment of Circulating MicroRNAs for the Diagnosis and Disease Activity Evaluation in Patients with Ulcerative Colitis by Using the Nanostring Technology
- (2015) Christos Polytarchou et al. INFLAMMATORY BOWEL DISEASES
- Curcumin suppresses NLRP3 inflammasome activation and protects against LPS-induced septic shock
- (2015) Zizhen Gong et al. MOLECULAR NUTRITION & FOOD RESEARCH
- Epithelial-derived IL-18 regulates Th17 cell differentiation and Foxp3+ Treg cell function in the intestine
- (2015) O J Harrison et al. Mucosal Immunology
- The receptor NLRP3 is a transcriptional regulator of TH2 differentiation
- (2015) Mélanie Bruchard et al. NATURE IMMUNOLOGY
- Role of the Microbiota in Immunity and Inflammation
- (2014) Yasmine Belkaid et al. CELL
- NLRP3 gene is associated with ulcerative colitis (UC), but not Crohn’s disease (CD), in Chinese Han population
- (2014) Hong-xin Zhang et al. INFLAMMATION RESEARCH
- Inflammasome activation has an important role in the development of spontaneous colitis
- (2014) J Zhang et al. Mucosal Immunology
- Inflammatory bowel disease: Pathogenesis
- (2014) Yi-Zhen Zhang WORLD JOURNAL OF GASTROENTEROLOGY
- New Insights into Mechanisms Controlling the NLRP3 Inflammasome and Its Role in Lung Disease
- (2013) Dominic De Nardo et al. AMERICAN JOURNAL OF PATHOLOGY
- The NLRP3 Inflammasome as a Novel Player of the Intercellular Crosstalk in Metabolic Disorders
- (2013) Elisa Benetti et al. MEDIATORS OF INFLAMMATION
- IL-1 and T Helper Immune Responses
- (2013) Veronica Santarlasci et al. Frontiers in Immunology
- New Insights in the Immunobiology of IL-1 Family Members
- (2013) Frank L. van de Veerdonk et al. Frontiers in Immunology
- Protective and Aggravating Effects of Nlrp3 Inflammasome Activation in IBD Models: Influence of Genetic and Environmental Factors
- (2012) Christian Bauer et al. DIGESTIVE DISEASES
- IL-1β mediates chronic intestinal inflammation by promoting the accumulation of IL-17A secreting innate lymphoid cells and CD4+Th17 cells
- (2012) Margherita Coccia et al. JOURNAL OF EXPERIMENTAL MEDICINE
- NLRP3 Inflammasome Activity Is Negatively Controlled by miR-223
- (2012) F. Bauernfeind et al. JOURNAL OF IMMUNOLOGY
- Dendritic Cells: Arbiters of Immunity and Immunological Tolerance
- (2012) K. L. Lewis et al. Cold Spring Harbor Perspectives in Biology
- A clinical perspective of IL-1β as the gatekeeper of inflammation
- (2011) Charles A. Dinarello EUROPEAN JOURNAL OF IMMUNOLOGY
- The Nlrp3 inflammasome: contributions to intestinal homeostasis
- (2011) Md. Hasan Zaki et al. TRENDS IN IMMUNOLOGY
- The Role of CD103+ Dendritic Cells in the Intestinal Mucosal Immune System
- (2011) Darren Thomas Ruane et al. Frontiers in Immunology
- Interleukin-18 in Intestinal Inflammation: Friend and Foe?
- (2010) Britta Siegmund IMMUNITY
- The NLRP3 Inflammasome Protects against Loss of Epithelial Integrity and Mortality during Experimental Colitis
- (2010) Md. Hasan Zaki et al. IMMUNITY
- Control of Intestinal Homeostasis, Colitis, and Colitis-Associated Colorectal Cancer by the Inflammatory Caspases
- (2010) Jeremy Dupaul-Chicoine et al. IMMUNITY
- Genetic association between NLRP3 variants and Crohnʼs disease does not replicate in a large UK panel
- (2010) Gregory J. Lewis et al. INFLAMMATORY BOWEL DISEASES
- NLRP3 inflammasome plays a key role in the regulation of intestinal homeostasis
- (2010) Simon A. Hirota et al. INFLAMMATORY BOWEL DISEASES
- Combined Polymorphisms in Genes Encoding the Inflammasome Components NALP3 and CARD8 Confer Susceptibility to Crohn's Disease in Swedish Men
- (2009) Ida Schoultz et al. AMERICAN JOURNAL OF GASTROENTEROLOGY
- Twin studies reveal specific imbalances in the mucosa-associated microbiota of patients with ileal Crohnʼs disease
- (2008) Ben Willing et al. INFLAMMATORY BOWEL DISEASES
- Common variants in the NLRP3 region contribute to Crohn's disease susceptibility
- (2008) Alexandra-Chloé Villani et al. NATURE GENETICS
- Local and systemic interleukin-18 and interleukin-18-binding protein in children with inflammatory bowel disease
- (2007) Steven T. Leach et al. INFLAMMATORY BOWEL DISEASES
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowCreate your own webinar
Interested in hosting your own webinar? Check the schedule and propose your idea to the Peeref Content Team.
Create Now