Hormetic potential of methylglyoxal, a side-product of glycolysis, in switching tumours from growth to death
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Title
Hormetic potential of methylglyoxal, a side-product of glycolysis, in switching tumours from growth to death
Authors
Keywords
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Journal
Scientific Reports
Volume 7, Issue 1, Pages -
Publisher
Springer Nature
Online
2017-09-11
DOI
10.1038/s41598-017-12119-7
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Note: Only part of the references are listed.- Methylglyoxal-derived stress: An emerging biological factor involved in the onset and progression of cancer
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- (2016) Naila Rabbani et al. GLYCOCONJUGATE JOURNAL
- Loss of Glyoxalase 1 Induces Compensatory Mechanism to Achieve Dicarbonyl Detoxification in Mammalian Schwann Cells
- (2016) Jakob Morgenstern et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- The Warburg Effect: How Does it Benefit Cancer Cells?
- (2016) Maria V. Liberti et al. TRENDS IN BIOCHEMICAL SCIENCES
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- (2016) Silvia Menegon et al. TRENDS IN MOLECULAR MEDICINE
- Methylglyoxal, a glycolysis side-product, induces Hsp90 glycation and YAP-mediated tumor growth and metastasis
- (2016) Marie-Julie Nokin et al. eLife
- Aldo-keto reductase 1C3 (AKR1C3) is associated with the doxorubicin resistance in human breast cancer via PTEN Loss
- (2015) Ting Zhong et al. BIOMEDICINE & PHARMACOTHERAPY
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- (2015) Dionne E.M. Maessen et al. CLINICAL SCIENCE
- Triple negative tumors accumulate significantly less methylglyoxal specific adducts than other human breast cancer subtypes
- (2015) Barbara Chiavarina et al. Oncotarget
- Long-term In Vitro Treatment of Human Glioblastoma Cells with Temozolomide Increases Resistance In Vivo through Up-regulation of GLUT Transporter and Aldo-Keto Reductase Enzyme AKR1C Expression
- (2015) Benjamin Le Calvé et al. NEOPLASIA
- LR-90 prevents methylglyoxal-induced oxidative stress and apoptosis in human endothelial cells
- (2014) James L. Figarola et al. APOPTOSIS
- Glycoxidation of biological macromolecules: A critical approach to halt the menace of glycation
- (2014) S. Ahmad et al. GLYCOBIOLOGY
- Measurement of methylglyoxal by stable isotopic dilution analysis LC-MS/MS with corroborative prediction in physiological samples
- (2014) Naila Rabbani et al. Nature Protocols
- Integrated genomic and functional analyses reveal glyoxalase I as a novel metabolic oncogene in human gastric cancer
- (2014) F Hosoda et al. ONCOGENE
- The Anti-Proliferative Effect of L-Carnosine Correlates with a Decreased Expression of Hypoxia Inducible Factor 1 alpha in Human Colon Cancer Cells
- (2014) Barbara Iovine et al. PLoS One
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- (2014) Yao Shen et al. PLoS One
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- (2013) Xiaohui Hu et al. BIOTECHNOLOGY LETTERS
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- (2013) Kyeong-Ah Jung et al. TOXICOLOGY LETTERS
- Metabolic Reprogramming: A Cancer Hallmark Even Warburg Did Not Anticipate
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- Transcriptional control of glyoxalase 1 by Nrf2 provides a stress-responsive defence against dicarbonyl glycation
- (2011) Mingzhan Xue et al. BIOCHEMICAL JOURNAL
- GLO1-A novel amplified gene in human cancer
- (2010) Thomas Santarius et al. GENES CHROMOSOMES & CANCER
- GLO1 overexpression in human malignant melanoma
- (2010) Warner B. Bair et al. MELANOMA RESEARCH
- Hyperglycemia-Induced Reactive Oxygen Species Increase Expression of the Receptor for Advanced Glycation End Products (RAGE) and RAGE Ligands
- (2009) D. Yao et al. DIABETES
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