Fatty acid and endotoxin activate inflammasomes in mouse hepatocytes that release danger signals to stimulate immune cells
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Title
Fatty acid and endotoxin activate inflammasomes in mouse hepatocytes that release danger signals to stimulate immune cells
Authors
Keywords
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Journal
HEPATOLOGY
Volume 54, Issue 1, Pages 133-144
Publisher
Wiley
Online
2011-04-12
DOI
10.1002/hep.24341
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- The Inflammasomes: Guardians of the Body
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- Characterization of High-Fat, Diet-Induced, Non-alcoholic Steatohepatitis with Fibrosis in Rats
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- Increased intestinal permeability and tight junction alterations in nonalcoholic fatty liver disease
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- Toll-like receptor 4 is a key mediator of murine steatotic liver warm ischemia/reperfusion injury
- (2009) Justin D. Ellett et al. LIVER TRANSPLANTATION
- High Fat Feeding Induces Hepatic Fatty Acid Elongation in Mice
- (2009) Maaike H. Oosterveer et al. PLoS One
- Lipotoxicity in nonalcoholic fatty liver disease: not all lipids are created equal
- (2009) Naim Alkhouri et al. Expert Review of Gastroenterology & Hepatology
- Differential requirement for the activation of the inflammasome for processing and release of IL-1 in monocytes and macrophages
- (2008) M. G. Netea et al. BLOOD
- The Caspase-1 Inflammasome: A Pilot of Innate Immune Responses
- (2008) Hong Bing Yu et al. Cell Host & Microbe
- Beyond insulin resistance: Innate immunity in nonalcoholic steatohepatitis
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- Nonalcoholic fatty liver disease and nonalcoholic steatohepatitis: Selected practical issues in their evaluation and management
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- Stimulation of Toll-like receptor 3 and 4 induces interleukin-1β maturation by caspase-8
- (2008) Jonathan Maelfait et al. JOURNAL OF EXPERIMENTAL MEDICINE
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