Mitochondria at the heart of cardiovascular protection: p66shc—friend or foe?
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Title
Mitochondria at the heart of cardiovascular protection: p66shc—friend or foe?
Authors
Keywords
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Journal
EUROPEAN HEART JOURNAL
Volume 36, Issue 8, Pages 469-471
Publisher
Oxford University Press (OUP)
Online
2014-10-22
DOI
10.1093/eurheartj/ehu409
References
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Note: Only part of the references are listed.- Genetic deletion of the adaptor protein p66Shc increases susceptibility to short-term ischaemic myocardial injury via intracellular salvage pathways
- (2014) Alexander Akhmedov et al. EUROPEAN HEART JOURNAL
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- (2014) K. N. Alavian et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- The STAT3 Inhibitor Stattic Impairs Cardiomyocyte Mitochondrial Function Through Increased Reactive Oxygen Species Formation
- (2013) Kerstin Boengler et al. CURRENT PHARMACEUTICAL DESIGN
- Connexin 43 impacts on mitochondrial potassium uptake
- (2013) Kerstin Boengler et al. Frontiers in Pharmacology
- Gene Silencing of the Mitochondrial Adaptor p66 Shc Suppresses Vascular Hyperglycemic Memory in Diabetes
- (2012) Francesco Paneni et al. CIRCULATION RESEARCH
- Deletion of the ageing gene p66Shc reduces early stroke size following ischaemia/reperfusion brain injury
- (2012) Remo D. Spescha et al. EUROPEAN HEART JOURNAL
- Cardioprotection: chances and challenges of its translation to the clinic
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- The p66Shcknockout mice are short lived under natural condition
- (2011) Marco Giorgio et al. AGING CELL
- Mitochondrial STAT3 Activation and Cardioprotection by Ischemic Postconditioning in Pigs With Regional Myocardial Ischemia/Reperfusion
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- Inhibition of mitochondrial permeability transition pore opening: the holy grail of cardioprotection
- (2010) Gerd Heusch et al. BASIC RESEARCH IN CARDIOLOGY
- Inhibition of permeability transition pore opening by mitochondrial STAT3 and its role in myocardial ischemia/reperfusion
- (2010) Kerstin Boengler et al. BASIC RESEARCH IN CARDIOLOGY
- Greater resistance to inflammation at adulthood could contribute to extended life span of p66Shc−/− mice
- (2010) Alessandra Berry et al. EXPERIMENTAL GERONTOLOGY
- Mitochondrial pathways for ROS formation and myocardial injury: the relevance of p66Shc and monoamine oxidase
- (2009) Fabio Di Lisa et al. BASIC RESEARCH IN CARDIOLOGY
- The cardioprotective effects elicited by p66Shc ablation demonstrate the crucial role of mitochondrial ROS formation in ischemia/reperfusion injury
- (2009) Andrea Carpi et al. BIOCHIMICA ET BIOPHYSICA ACTA-BIOENERGETICS
- Multiple protective pathways against reperfusion injury: A SAFE path without Aktion?
- (2009) Sandrine Lecour JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
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- (2008) Gerd Heusch et al. CIRCULATION
- Final Common Molecular Pathways of Aging and Cardiovascular Disease
- (2007) Francesco Cosentino et al. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
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