The nod-like receptor, Nlrp12, plays an anti-inflammatory role in experimental autoimmune encephalomyelitis
Published 2015 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
The nod-like receptor, Nlrp12, plays an anti-inflammatory role in experimental autoimmune encephalomyelitis
Authors
Keywords
<em class=EmphasisTypeItalic >Nlrp12</em>, Experimental autoimmune encephalomyelitis, Microglia, Neuroinflammations
Journal
Journal of Neuroinflammation
Volume 12, Issue 1, Pages -
Publisher
Springer Nature
Online
2015-10-31
DOI
10.1186/s12974-015-0414-5
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- The NLRP12 Sensor Negatively Regulates Autoinflammatory Disease by Modulating Interleukin-4 Production in T Cells
- (2015) John R. Lukens et al. IMMUNITY
- Role of the innate and adaptive immune responses in the course of multiple sclerosis
- (2015) Bernhard Hemmer et al. LANCET NEUROLOGY
- The multifaceted nature of NLRP12
- (2014) Sinem Tuncer et al. JOURNAL OF LEUKOCYTE BIOLOGY
- Initial Immunopathogenesis of Multiple Sclerosis: Innate Immune Response
- (2013) Norma Y. Hernández-Pedro et al. Clinical & Developmental Immunology
- The Nucleotide-binding Leucine-rich Repeat (NLR) Family Member NLRX1 Mediates Protection against Experimental Autoimmune Encephalomyelitis and Represses Macrophage/Microglia-induced Inflammation
- (2013) Timothy K. Eitas et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- NF-κB as a Target for Modulating Inflammatory Responses
- (2012) Chiara Gasparini et al. CURRENT PHARMACEUTICAL DESIGN
- NLRP12 Suppresses Colon Inflammation and Tumorigenesis through the Negative Regulation of Noncanonical NF-κB Signaling
- (2012) Irving C. Allen et al. IMMUNITY
- The biology of chemokines and their receptors
- (2012) Sergio A. Lira et al. IMMUNOLOGIC RESEARCH
- NLRP3 inflammasome induces chemotactic immune cell migration to the CNS in experimental autoimmune encephalomyelitis
- (2012) M. Inoue et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Signaling in Innate Immunity and Inflammation
- (2012) K. Newton et al. Cold Spring Harbor Perspectives in Biology
- Experimental autoimmune encephalomyelitis (EAE) as a model for multiple sclerosis (MS)
- (2011) Cris S Constantinescu et al. BRITISH JOURNAL OF PHARMACOLOGY
- The NOD-Like Receptor NLRP12 Attenuates Colon Inflammation and Tumorigenesis
- (2011) Md. Hasan Zaki et al. CANCER CELL
- How do immune cells overcome the blood-brain barrier in multiple sclerosis?
- (2011) Catherine Larochelle et al. FEBS LETTERS
- Pathogen Recognition by the Innate Immune System
- (2011) Himanshu Kumar et al. INTERNATIONAL REVIEWS OF IMMUNOLOGY
- NLR functions beyond pathogen recognition
- (2011) Thomas A Kufer et al. NATURE IMMUNOLOGY
- Repertoire of microglial and macrophage responses after spinal cord injury
- (2011) Samuel David et al. NATURE REVIEWS NEUROSCIENCE
- Cutting Edge: NLRP12 Controls Dendritic and Myeloid Cell Migration To Affect Contact Hypersensitivity
- (2010) J. C. Arthur et al. JOURNAL OF IMMUNOLOGY
- NLRP3 Plays a Critical Role in the Development of Experimental Autoimmune Encephalomyelitis by Mediating Th1 and Th17 Responses
- (2010) D. Gris et al. JOURNAL OF IMMUNOLOGY
- The Inflammasome Sensor, NLRP3, Regulates CNS Inflammation and Demyelination via Caspase-1 and Interleukin-18
- (2010) S. Jha et al. JOURNAL OF NEUROSCIENCE
- The chemokine receptor CCR5 in the central nervous system
- (2010) Silvia Sorce et al. PROGRESS IN NEUROBIOLOGY
- The Nuclear Factor NF- B Pathway in Inflammation
- (2009) T. Lawrence Cold Spring Harbor Perspectives in Biology
- Analyzing real-time PCR data by the comparative CT method
- (2008) Thomas D Schmittgen et al. Nature Protocols
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started