Cancer-associated fibroblasts induce high mobility group box 1 and contribute to resistance to doxorubicin in breast cancer cells
Published 2015 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Cancer-associated fibroblasts induce high mobility group box 1 and contribute to resistance to doxorubicin in breast cancer cells
Authors
Keywords
Breast cancer, Cancer-associated fibroblast, HMGB1, Chemoresistance
Journal
BMC CANCER
Volume 14, Issue 1, Pages -
Publisher
Springer Nature
Online
2015-06-18
DOI
10.1186/1471-2407-14-955
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Autophagy Inhibition Augments the Anticancer Effects of Epirubicin Treatment in Anthracycline-Sensitive and -Resistant Triple-Negative Breast Cancer
- (2014) S. Chittaranjan et al. CLINICAL CANCER RESEARCH
- Fibroblasts Protect Melanoma Cells from the Cytotoxic Effects of Doxorubicin
- (2014) Manoela Tiago et al. TISSUE ENGINEERING PART A
- Serum HMGB1 as a prognostic marker for malignant pleural mesothelioma
- (2013) Chiharu Tabata et al. BMC CANCER
- Ovarian cancer microenvironment: implications for cancer dissemination and chemoresistance acquisition
- (2013) Benoît Thibault et al. CANCER AND METASTASIS REVIEWS
- Breast stromal fibroblasts from histologically normal surgical margins are pro-carcinogenic
- (2013) Maha A Al-Rakan et al. JOURNAL OF PATHOLOGY
- Adjuvant Systemic Therapies in Breast Cancer
- (2013) Leonel F. Hernandez-Aya et al. SURGICAL CLINICS OF NORTH AMERICA
- The Role of Glycyrrhizin, an Inhibitor of HMGB1 Protein, in Anticancer Therapy
- (2012) Ryszard Smolarczyk et al. ARCHIVUM IMMUNOLOGIAE ET THERAPIAE EXPERIMENTALIS
- Stromal cells in tumor microenvironment and breast cancer
- (2012) Yan Mao et al. CANCER AND METASTASIS REVIEWS
- Cancer Cell Secretion of the DAMP Protein HMGB1 Supports Progression in Malignant Mesothelioma
- (2012) S. Jube et al. CANCER RESEARCH
- The Tumor Stroma as Mediator of Drug Resistance - A Potential Target to Improve Cancer Therapy?
- (2012) Susanne Sebens et al. CURRENT PHARMACEUTICAL BIOTECHNOLOGY
- High mobility group box 1 released from necrotic cells enhances regrowth and metastasis of cancer cells that have survived chemotherapy
- (2012) Yi Luo et al. EUROPEAN JOURNAL OF CANCER
- High-mobility group box 1 activates caspase-1 and promotes hepatocellular carcinoma invasiveness and metastases
- (2012) Wei Yan et al. HEPATOLOGY
- Epithelial-mesenchymal transition in breast cancer correlates with high histological grade and triple-negative phenotype
- (2012) Hoiseon Jeong et al. HISTOPATHOLOGY
- Implication of Tumor Microenvironment in Chemoresistance: Tumor-Associated Stromal Cells Protect Tumor Cells from Cell Death
- (2012) Magali Castells et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
- HMGB1 is overexpressed in tumor cells and promotes activity of regulatory T cells in patients with head and neck cancer
- (2012) Clarissa A. Wild et al. ORAL ONCOLOGY
- Diagnostic Significance of Serum HMGB1 in Colorectal Carcinomas
- (2012) Hanna Lee et al. PLoS One
- Circulating immunogenic cell death biomarkers HMGB1 and RAGE in breast cancer patients during neoadjuvant chemotherapy
- (2012) Oliver J. Stoetzer et al. TUMOR BIOLOGY
- Global cancer statistics
- (2011) Ahmedin Jemal et al. CA-A CANCER JOURNAL FOR CLINICIANS
- Anti-estrogen resistance in breast cancer is induced by the tumor microenvironment and can be overcome by inhibiting mitochondrial function in epithelial cancer cells
- (2011) Ubaldo E. Martinez-Outschoorn et al. CANCER BIOLOGY & THERAPY
- TNF-α induced secretion of HMGB1 from non-immune canine mammary epithelial cells (MTH53A)
- (2011) Saskia Willenbrock et al. CYTOKINE
- DNA Alkylating Therapy Induces Tumor Regression through an HMGB1-Mediated Activation of Innate Immunity
- (2011) J. L. Guerriero et al. JOURNAL OF IMMUNOLOGY
- HMGB1 and RAGE in Inflammation and Cancer
- (2010) Gary P. Sims et al. Annual Review of Immunology
- DAMP-mediated autophagy contributes to drug resistance
- (2010) Liying Liu et al. Autophagy
- Hmgb1 Promotes Wound Healing of 3T3 Mouse Fibroblasts via Rage-Dependent ERK1/2 Activation
- (2010) Elia Ranzato et al. CELL BIOCHEMISTRY AND BIOPHYSICS
- HMGB-1 induces IL-6 production in human synovial fibroblasts through c-Src, Akt and NF-κB pathways
- (2010) Chun-Han Hou et al. JOURNAL OF CELLULAR PHYSIOLOGY
- HMGB1-induced autophagy promotes chemotherapy resistance in leukemia cells
- (2010) L Liu et al. LEUKEMIA
- Gene expression profiling of cholangiocarcinoma-derived fibroblast reveals alterations related to tumor progression and indicates periostin as a poor prognostic marker
- (2010) Kusumawadee Utispan et al. Molecular Cancer
- Cancer associated fibroblasts (CAFs) in tumor microenvironment
- (2009) Fei Xing Frontiers in Bioscience-Landmark
- The expression of HMGB1 protein and its receptor RAGE in human malignant tumors
- (2009) Nora Kostova et al. MOLECULAR AND CELLULAR BIOCHEMISTRY
- A stroma-related gene signature predicts resistance to neoadjuvant chemotherapy in breast cancer
- (2009) Pierre Farmer et al. NATURE MEDICINE
- Cancer associated fibroblasts in cancer pathogenesis
- (2009) Omar E. Franco et al. SEMINARS IN CELL & DEVELOPMENTAL BIOLOGY
- Epithelial-Mesenchymal Transition in Breast Cancer Relates to the Basal-like Phenotype
- (2008) D. Sarrio et al. CANCER RESEARCH
- Role of myofibroblasts in innate chemoresistance of pancreatic carcinoma-Epigenetic downregulation of caspases
- (2008) Susanne Sebens Müerköster et al. INTERNATIONAL JOURNAL OF CANCER
- Increased expression of high mobility group box 1 (HMGB1) is associated with progression and poor prognosis in human nasopharyngeal carcinoma
- (2008) D Wu et al. JOURNAL OF PATHOLOGY
- Non-Hodgkin lymphoma expressing high levels of the danger-signalling protein HMGB1
- (2008) Anke Meyer et al. LEUKEMIA & LYMPHOMA
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started