Proximal tubule-dominant transfer of AT1a receptors induces blood pressure responses to intracellular angiotensin II in AT1a receptor-deficient mice
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Title
Proximal tubule-dominant transfer of AT1a receptors induces blood pressure responses to intracellular angiotensin II in AT1a receptor-deficient mice
Authors
Keywords
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Journal
AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY
Volume 304, Issue 8, Pages R588-R598
Publisher
American Physiological Society
Online
2013-02-21
DOI
10.1152/ajpregu.00338.2012
References
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Note: Only part of the references are listed.- Renal proximal tubule angiotensin AT1A receptors regulate blood pressure
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- (2011) Susan B. Gurley et al. Cell Metabolism
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- Under pressure: the search for the essential mechanisms of hypertension
- (2011) Thomas M Coffman NATURE MEDICINE
- New insights and perspectives on intrarenal renin-angiotensin system: Focus on intracrine/intracellular angiotensin II
- (2011) Jia L. Zhuo et al. PEPTIDES
- Regulation of Blood Pressure and Salt Homeostasis by Endothelin
- (2011) Donald E. Kohan et al. PHYSIOLOGICAL REVIEWS
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- Transgenic mice expressing an intracellular fluorescent fusion of angiotensin II demonstrate renal thrombotic microangiopathy and elevated blood pressure
- (2010) K. M. Redding et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Polycystin-dependent fluid flow sensing targets histone deacetylase 5 to prevent the development of renal cysts
- (2010) S. Xia et al. DEVELOPMENT
- Intrarenal Angiotensin-Converting Enzyme Induces Hypertension in Response to Angiotensin I Infusion
- (2010) R. A. Gonzalez-Villalobos et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
- Angiotensin-(1-7)-Angiotensin-Converting Enzyme 2 Attenuates Reactive Oxygen Species Formation to Angiotensin II Within the Cell Nucleus
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- Intracellular ANG II directly induces in vitro transcription of TGF-β1, MCP-1, and NHE-3 mRNAs in isolated rat renal cortical nuclei via activation of nuclear AT1a receptors
- (2008) Xiao C. Li et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
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