ClC-3 is a candidate of the channel proteins mediating acid-activated chloride currents in nasopharyngeal carcinoma cells

Enhanced inhibitory effect of acidosis on hERG potassium channels that incorporate the hERG1b isoform

(2011) C.Y. Du et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS

Differential expression and roles of volume-activated chloride channels in control of growth of normal and cancerous nasopharyngeal epithelial cells

(2011) Linyan Zhu et al.   BIOCHEMICAL PHARMACOLOGY

ClC-3 is a main component of background chloride channels activated under isotonic conditions by autocrine ATP in nasopharyngeal carcinoma cells

(2010) Linjie Yang et al.   JOURNAL OF CELLULAR PHYSIOLOGY

Lack of association between stretch-activated and volume-activated Cl− currents in hepatocellular carcinoma cells

(2010) Jianwen Mao et al.   JOURNAL OF CELLULAR PHYSIOLOGY

Inhibition of KCa2.2 and KCa2.3 channel currents by protonation of outer pore histidine residues

(2009) Samuel J. Goodchild et al.   JOURNAL OF GENERAL PHYSIOLOGY

Extracellular acidification exerts opposite actions on TREK1 and TREK2 potassium channels via a single conserved histidine residue

(2009) G. Sandoz et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Volume-activated chloride channels contribute to cell-cycle-dependent regulation of HeLa cell migration

(2008) Jianwen Mao et al.   BIOCHEMICAL PHARMACOLOGY

Extracellular pH Modulates the Voltage-dependent Ca2+ Current and Low Threshold K+ Current in Hair Cells

(2008) Angélica Almanza et al.   NEUROCHEMICAL RESEARCH

Modulation of the Kir7.1 potassium channel by extracellular and intracellular pH

(2007) Bret A. Hughes et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY