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SnapShot: Talin and the Modular Nature of the Integrin Adhesome

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WAVE3-NFκB Interplay Is Essential for the Survival and Invasion of Cancer Cells

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miR-200b suppresses invasiveness and modulates the cytoskeletal and adhesive machinery in esophageal squamous cell carcinoma cells via targeting Kindlin-2

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Kindlin 2 promotes breast cancer invasion via epigenetic silencing of the microRNA200 gene family

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Kindlin 2 forms a transcriptional complex with β-catenin and TCF4 to enhance Wnt signalling

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Spatial Coordination of Kindlin-2 with Talin Head Domain in Interaction with Integrin β Cytoplasmic Tails

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miR-31 and its host gene lncRNA LOC554202 are regulated by promoter hypermethylation in triple-negative breast cancer

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The integrin coactivator Kindlin-2 plays a critical role in angiogenesis in mice and zebrafish

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miR-31 Is a Broad Regulator of  1-Integrin Expression and Function in Cancer Cells

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The emerging role of miR-200 family of MicroRNAs in epithelial-mesenchymal transition and cancer metastasis

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The Integrin Co-activator Kindlin-3 Is Expressed and Functional in a Non-hematopoietic Cell, the Endothelial Cell

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microRNAs and EMT in Mammary Cells and Breast Cancer

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The miR-200 Family: Central Player for Gain and Loss of the Epithelial Phenotype

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The miR200 Family of MicroRNAs Regulates WAVE3-dependent Cancer Cell Invasion

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Epithelial-Mesenchymal Transition: At the Crossroads of Development and Tumor Metastasis

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Kindlins: essential regulators of integrin signalling and cell–matrix adhesion

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A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

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The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

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The miR-200 Family Inhibits Epithelial-Mesenchymal Transition and Cancer Cell Migration by Direct Targeting of E-cadherin Transcriptional RepressorsZEB1andZEB2

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The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

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Non-coding RNAs take centre stage in epithelial-to-mesenchymal transition

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