Piezo1 is a mechanically activated ion channel and mediates pressure induced pancreatitis
Published 2018 View Full Article
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Title
Piezo1 is a mechanically activated ion channel and mediates pressure induced pancreatitis
Authors
Keywords
-
Journal
Nature Communications
Volume 9, Issue 1, Pages -
Publisher
Springer Nature
Online
2018-04-24
DOI
10.1038/s41467-018-04194-9
References
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Related references
Note: Only part of the references are listed.- Structure and mechanogating mechanism of the Piezo1 channel
- (2018) Qiancheng Zhao et al. NATURE
- Structure of the mechanically activated ion channel Piezo1
- (2017) Kei Saotome et al. NATURE
- Touch, Tension, and Transduction – The Function and Regulation of Piezo Ion Channels
- (2017) Jason Wu et al. TRENDS IN BIOCHEMICAL SCIENCES
- Prevention of Post–Endoscopic Retrograde Cholangiopancreatography Pancreatitis: Medications and Techniques
- (2016) Andrew Y. Wang et al. Clinical Gastroenterology and Hepatology
- Acute Pancreatitis
- (2016) Chris E. Forsmark et al. NEW ENGLAND JOURNAL OF MEDICINE
- Architecture of the mammalian mechanosensitive Piezo1 channel
- (2015) Jingpeng Ge et al. NATURE
- Structural similarities and differences between the human and the mouse pancreas
- (2015) Jurij Dolenšek et al. Islets
- Chemical activation of the mechanotransduction channel Piezo1
- (2015) Ruhma Syeda et al. eLife
- Piezo1 links mechanical forces to red blood cell volume
- (2015) Stuart M Cahalan et al. eLife
- Ionic Selectivity and Permeation Properties of Human PIEZO1 Channels
- (2015) Radhakrishnan Gnanasambandam et al. PLoS One
- Functional Role for Piezo1 in Stretch-evoked Ca2+Influx and ATP Release in Urothelial Cell Cultures
- (2014) Tatsuya Miyamoto et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Piezo1 integration of vascular architecture with physiological force
- (2014) Jing Li et al. NATURE
- Piezo channels: from structure to function
- (2014) Linda Volkers et al. PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY
- Piezo1, a mechanically activated ion channel, is required for vascular development in mice
- (2014) S. S. Ranade et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Tuning Piezo ion channels to detect molecular-scale movements relevant for fine touch
- (2014) Kate Poole et al. Nature Communications
- Spatiotemporal patterns of multipotentiality in Ptf1a-expressing cells during pancreas organogenesis and injury-induced facultative restoration
- (2013) F. C. Pan et al. DEVELOPMENT
- The role of Ca2+in the pathophysiology of pancreatitis
- (2013) Julia V. Gerasimenko et al. JOURNAL OF PHYSIOLOGY-LONDON
- Ca2+ release-activated Ca2+ channel blockade as a potential tool in antipancreatitis therapy
- (2013) J. V. Gerasimenko et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Piezo1
- (2012) Philip A. Gottlieb et al. Channels
- How We Feel: Ion Channel Partnerships that Detect Mechanical Inputs and Give Rise to Touch and Pain Perception
- (2012) Shana L. Geffeney et al. NEURON
- The Mechanosensitive Ion Channel Piezo1 Is Inhibited by the Peptide GsMTx4
- (2011) Chilman Bae et al. BIOCHEMISTRY
- Molecular mechanisms of mechanotransduction in mammalian sensory neurons
- (2011) Patrick Delmas et al. NATURE REVIEWS NEUROSCIENCE
- Canonical TRP channels and mechanotransduction: from physiology to disease states
- (2010) Amanda Patel et al. PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY
- Piezo1 and Piezo2 Are Essential Components of Distinct Mechanically Activated Cation Channels
- (2010) B. Coste et al. SCIENCE
- Early Endoscopic Retrograde Cholangiopancreatography in Predicted Severe Acute Biliary Pancreatitis
- (2009) Hjalmar C. van Santvoort et al. ANNALS OF SURGERY
- Fatty acids, alcohol and fatty acid ethyl esters: Toxic Ca2+ signal generation and pancreatitis
- (2009) O.H. Petersen et al. CELL CALCIUM
- An Improved Method for Extracting Myeloperoxidase and Determining Its Activity in the Pancreas and Lungs During Pancreatitis
- (2009) Rajinder Dawra et al. PANCREAS
- Ca2+-induced pancreatic cell death: Roles of the endoplasmic reticulum, zymogen granules, lysosomes and endosomes
- (2008) Ole H Petersen JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY
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