Podocyte Activation of NLRP3 Inflammasomes Contributes to the Development of Proteinuria in Lupus Nephritis
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Title
Podocyte Activation of NLRP3 Inflammasomes Contributes to the Development of Proteinuria in Lupus Nephritis
Authors
Keywords
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Journal
Arthritis & Rheumatology
Volume 69, Issue 8, Pages 1636-1646
Publisher
Wiley
Online
2017-05-24
DOI
10.1002/art.40155
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Note: Only part of the references are listed.- Mitochondrial function is required for extracellular ATP-induced NLRP3 inflammasome activation
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- Podocyte injury and repair mechanisms
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- Role of NLRP3 Inflammasomes for Rhabdomyolysis-induced Acute Kidney Injury
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- Lupus Nephritis: Glycogen Synthase Kinase 3β Promotion of Renal Damage Through Activation of the NLRP3 Inflammasome in Lupus-Prone Mice
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- Mechanisms and Functions of Inflammasomes
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- Genetics of systemic lupus erythematosus: immune responses and end organ resistance to damage
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- Podocyte involvement in lupus nephritis based on the 2003 ISN/RPS system: a large cohort study from a single centre
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- P2X7Blockade Attenuates Murine Lupus Nephritis by Inhibiting Activation of the NLRP3/ASC/Caspase 1 Pathway
- (2013) Jijun Zhao et al. ARTHRITIS AND RHEUMATISM
- Cgnz1allele confers kidney resistance to damage preventing progression of immune complex–mediated acute lupus glomerulonephritis
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- Podocyte injury in pure membranous and proliferative lupus nephritis: distinct underlying mechanisms of proteinuria?
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- B-cell depletion in the treatment of lupus nephritis
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- Long-term mortality and renal outcome in a cohort of 100 patients with Lupus Nephritis
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