Absence of microRNA-21 does not reduce muscular dystrophy in mouse models of LAMA2-CMD

Chimeric protein repair of laminin polymerization ameliorates muscular dystrophy phenotype

(2017) Karen K. McKee et al.   JOURNAL OF CLINICAL INVESTIGATION

Potent pro-inflammatory and pro-fibrotic molecules, osteopontin and galectin-3, are not major disease modulators of laminin α2 chain-deficient muscular dystrophy

(2017) Kinga I. Gawlik et al.   Scientific Reports

Opposing roles of miR-21 and miR-29 in the progression of fibrosis in Duchenne muscular dystrophy

(2015) Simona Zanotti et al.   BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE

Osteopontin is indispensible for AP1-mediated angiotensin II-related miR-21 transcription during cardiac fibrosis

(2015) Johan M. Lorenzen et al.   EUROPEAN HEART JOURNAL

Loss of Dystrophin and β-Sarcoglycan Significantly Exacerbates the Phenotype of Laminin α2 Chain–Deficient Animals

(2014) Kinga I. Gawlik et al.   AMERICAN JOURNAL OF PATHOLOGY

DIANA-TarBase v7.0: indexing more than half a million experimentally supported miRNA:mRNA interactions

(2014) Ioannis S. Vlachos et al.   NUCLEIC ACIDS RESEARCH

Laminin α2 Chain-Deficiency is Associated with microRNA Deregulation in Skeletal Muscle and Plasma

(2014) Johan Holmberg et al.   Frontiers in Aging Neuroscience

Restoration of muscle strength in dystrophic muscle by angiotensin-1-7 through inhibition of TGF-β signalling

(2013) María José Acuña et al.   HUMAN MOLECULAR GENETICS

Laminin-211 in skeletal muscle function

(2013) Johan Holmberg et al.   Cell Adhesion & Migration

Laminin-111 Protein Therapy Reduces Muscle Pathology and Improves Viability of a Mouse Model of Merosin-Deficient Congenital Muscular Dystrophy

(2012) Jachinta E. Rooney et al.   AMERICAN JOURNAL OF PATHOLOGY

Cellular Mechanisms of Tissue Fibrosis. 1. Common and organ-specific mechanisms associated with tissue fibrosis

(2012) Michael Zeisberg et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

MKP-1 coordinates ordered macrophage-phenotype transitions essential for stem cell-dependent tissue repair

(2012) Eusebio Perdiguero et al.   CELL CYCLE

PAI-1–regulated miR-21 defines a novel age-associated fibrogenic pathway in muscular dystrophy

(2012) Esther Ardite et al.   JOURNAL OF CELL BIOLOGY

Angiotensin II type 1 receptor antagonists alleviate muscle pathology in the mouse model for laminin-α2-deficient congenital muscular dystrophy (MDC1A)

(2012) Sarina Meinen et al.   Skeletal Muscle

Losartan, a therapeutic candidate in congenital muscular dystrophy: Studies in the dy2J/dy2J Mouse

(2011) Moran Elbaz et al.   ANNALS OF NEUROLOGY

Muscle-specific expression of insulin-like growth factor 1 improves outcome in Lama2Dy-w mice, a model for congenital muscular dystrophy type 1A

(2011) A. Kumar et al.   HUMAN MOLECULAR GENETICS

Autophagy is increased in laminin α2 chain-deficient muscle and its inhibition improves muscle morphology in a mouse model of MDC1A

(2011) Virginie Carmignac et al.   HUMAN MOLECULAR GENETICS

p38/MKP-1–regulated AKT coordinates macrophage transitions and resolution of inflammation during tissue repair

(2011) Eusebio Perdiguero et al.   JOURNAL OF CELL BIOLOGY

Loss of the miR-21 allele elevates the expression of its target genes and reduces tumorigenesis

(2011) X. Ma et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Skeletal muscle laminin and MDC1A: pathogenesis and treatment strategies

(2011) Kinga I Gawlik et al.   Skeletal Muscle

Proteasome inhibition improves the muscle of laminin α2 chain-deficient mice

(2010) Virginie Carmignac et al.   HUMAN MOLECULAR GENETICS

Transgenic overexpression of laminin α1 chain in laminin α2 chain-deficient mice rescues the disease throughout the lifespan

(2010) Kinga I. Gawlik et al.   MUSCLE & NERVE

Omigapil Ameliorates the Pathology of Muscle Dystrophy Caused by Laminin- 2 Deficiency

(2009) M. Erb et al.   JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS

MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts

(2008) Thomas Thum et al.   NATURE