Transgenic Mice Overexpressing the Divalent Metal Transporter 1 Exhibit Iron Accumulation and Enhanced Parkin Expression in the Brain
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Title
Transgenic Mice Overexpressing the Divalent Metal Transporter 1 Exhibit Iron Accumulation and Enhanced Parkin Expression in the Brain
Authors
Keywords
6-OHDA, Dopamine, Oxidative stress, Parkin, Parkinson’s disease
Journal
NEUROMOLECULAR MEDICINE
Volume 19, Issue 2-3, Pages 375-386
Publisher
Springer Nature
Online
2017-07-10
DOI
10.1007/s12017-017-8451-0
References
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Related references
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- Iron Accumulation Is Not Homogenous among Patients with Parkinson’s Disease
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- Substrate Profile and Metal-ion Selectivity of Human Divalent Metal-ion Transporter-1
- (2012) Anthony C. Illing et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- The role of iron in neurodegeneration—Mössbauer spectroscopy, electron microscopy, enzyme-linked immunosorbent assay and neuroimaging studies
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- Parkin regulates metal transport via proteasomal degradation of the 1B isoforms of divalent metal transporter 1
- (2010) Jerome A. Roth et al. JOURNAL OF NEUROCHEMISTRY
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- (2009) Michael D. Garrick et al. BIOCHIMICA ET BIOPHYSICA ACTA-GENERAL SUBJECTS
- Absence of nigral degeneration in aged parkin/DJ-1/PINK1 triple knockout mice
- (2009) Tohru Kitada et al. JOURNAL OF NEUROCHEMISTRY
- Divalent metal transporter 1 (DMT1) contributes to neurodegeneration in animal models of Parkinson's disease
- (2008) J. Salazar et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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