Acutely damaged axons are remyelinated in multiple sclerosis and experimental models of demyelination
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Title
Acutely damaged axons are remyelinated in multiple sclerosis and experimental models of demyelination
Authors
Keywords
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Journal
GLIA
Volume 65, Issue 8, Pages 1350-1360
Publisher
Wiley
Online
2017-05-31
DOI
10.1002/glia.23167
References
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Related references
Note: Only part of the references are listed.- Relationship of acute axonal damage, Wallerian degeneration, and clinical disability in multiple sclerosis
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- Pervasive Axonal Transport Deficits in Multiple Sclerosis Models
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- Astrocytes regulate myelin clearance through recruitment of microglia during cuprizone-induced demyelination
- (2012) Thomas Skripuletz et al. BRAIN
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- Oligodendroglia metabolically support axons and contribute to neurodegeneration
- (2012) Youngjin Lee et al. NATURE
- Late motor decline after accomplished remyelination: Impact for progressive multiple sclerosis
- (2011) Natalia Manrique-Hoyos et al. ANNALS OF NEUROLOGY
- A reversible form of axon damage in experimental autoimmune encephalomyelitis and multiple sclerosis
- (2011) Ivana Nikić et al. NATURE MEDICINE
- Review: The architecture of inflammatory demyelinating lesions: implications for studies on pathogenesis
- (2011) H. Lassmann NEUROPATHOLOGY AND APPLIED NEUROBIOLOGY
- Regional differences between grey and white matter in cuprizone induced demyelination
- (2009) Viktoria Gudi et al. BRAIN RESEARCH
- Chronic toxic demyelination in the central nervous system leads to axonal damage despite remyelination
- (2009) Maren Lindner et al. NEUROSCIENCE LETTERS
- Remyelination protects axons from demyelination-associated axon degeneration
- (2008) K. A. Irvine et al. BRAIN
- MHC class II exacerbates demyelination in vivo independently of T cells
- (2008) Meenaxi M. Hiremath et al. JOURNAL OF NEUROIMMUNOLOGY
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