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Epigenetic mechanisms in diabetic complications and metabolic memory

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Histone Methylation Patterns Are Cell-Type Specific in Human Monocytes and Lymphocytes and Well Maintained at Core Genes

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(2014) Mitsuo Kato et al.   Nature Reviews Nephrology

Vitamin D Receptor Inhibits Nuclear Factor κB Activation by Interacting with IκB Kinase β Protein

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Epigenetic Histone Methylation Modulates Fibrotic Gene Expression

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BTBR Ob/Ob Mutant Mice Model Progressive Diabetic Nephropathy

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Epigenetic phenomena linked to diabetic complications

(2010) Luciano Pirola et al.   Nature Reviews Endocrinology

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Hyperglycemia Induces a Dynamic Cooperativity of Histone Methylase and Demethylase Enzymes Associated With Gene-Activating Epigenetic Marks That Coexist on the Lysine Tail

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ATP-Citrate Lyase Links Cellular Metabolism to Histone Acetylation

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Lymphocytes From Patients With Type 1 Diabetes Display a Distinct Profile of Chromatin Histone H3 Lysine 9 Dimethylation: An Epigenetic Study in Diabetes

(2008) F. Miao et al.   DIABETES

Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemia

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Glucose and GLP-1 Stimulate cAMP Production via Distinct Adenylyl Cyclases in INS-1E Insulinoma Cells

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Analyzing real-time PCR data by the comparative CT method

(2008) Thomas D Schmittgen et al.   Nature Protocols

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Renoprotective role of the vitamin D receptor in diabetic nephropathy

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