Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite
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Title
Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite
Authors
Keywords
Snord115, Prader-Willi syndrome, Serotonin 2C recptor, Alternate splicing, Feeding
Journal
Molecular Brain
Volume 9, Issue 1, Pages -
Publisher
Springer Nature
Online
2016-12-08
DOI
10.1186/s13041-016-0277-4
References
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