Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite

标题
Increased alternate splicing of Htr2c in a mouse model for Prader-Willi syndrome leads disruption of 5HT2C receptor mediated appetite
作者
关键词
Snord115, Prader-Willi syndrome, Serotonin 2C recptor, Alternate splicing, Feeding
出版物
Molecular Brain
Volume 9, Issue 1, Pages -
出版商
Springer Nature
发表日期
2016-12-08
DOI
10.1186/s13041-016-0277-4

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