Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression

Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells

(2016) Jens S. Kalchschmidt et al.   JOURNAL OF EXPERIMENTAL MEDICINE

RUNXsuper-enhancer control through the Notch pathway by Epstein-Barr virus transcription factors regulates B cell growth

(2016) Andrea Gunnell et al.   NUCLEIC ACIDS RESEARCH

EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells

(2016) Jens S. Kalchschmidt et al.   PLoS Pathogens

EBNA2 Drives Formation of New Chromosome Binding Sites and Target Genes for B-Cell Master Regulatory Transcription Factors RBP-jκ and EBF1

(2016) Fang Lu et al.   PLoS Pathogens

Integrative analysis of 111 reference human epigenomes

(2015) Anshul Kundaje et al.   NATURE

Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2

(2015) Quentin Bazot et al.   PLoS Pathogens

The EBNA3 Family of Epstein-Barr Virus Nuclear Proteins Associates with the USP46/USP12 Deubiquitination Complexes to Regulate Lymphoblastoid Cell Line Growth

(2015) Makoto Ohashi et al.   PLoS Pathogens

The UCSC Genome Browser database: 2015 update

(2014) Kate R. Rosenbloom et al.   NUCLEIC ACIDS RESEARCH

The pathogenesis of Epstein–Barr virus persistent infection

(2013) David A Thorley-Lawson et al.   Current Opinion in Virology

Inactivation of Intergenic Enhancers by EBNA3A Initiates and Maintains Polycomb Signatures across a Chromatin Domain Encoding CXCL10 and CXCL9

(2013) Marie L. Harth-Hertle et al.   PLoS Pathogens

The EBV Latent Antigen 3C Inhibits Apoptosis through Targeted Regulation of Interferon Regulatory Factors 4 and 8

(2013) Shuvomoy Banerjee et al.   PLoS Pathogens

Modulation of Enhancer Looping and Differential Gene Targeting by Epstein-Barr Virus Transcription Factors Directs Cellular Reprogramming

(2013) Michael J. McClellan et al.   PLoS Pathogens

Induction of p16INK4a Is the Major Barrier to Proliferation when Epstein-Barr Virus (EBV) Transforms Primary B Cells into Lymphoblastoid Cell Lines

(2013) Lenka Skalska et al.   PLoS Pathogens

The DNA damage response in viral-induced cellular transformation

(2012) P A Nikitin et al.   BRITISH JOURNAL OF CANCER

PR-Set7 and H4K20me1: at the crossroads of genome integrity, cell cycle, chromosome condensation, and transcription

(2012) D. B. Beck et al.   GENES & DEVELOPMENT

EBNA3B-deficient EBV promotes B cell lymphomagenesis in humanized mice and is found in human tumors

(2012) Robert E. White et al.   JOURNAL OF CLINICAL INVESTIGATION

Different Patterns of Epstein-Barr Virus Latency in Endemic Burkitt Lymphoma (BL) Lead to Distinct Variants within the BL-Associated Gene Expression Signature

(2012) G. L. Kelly et al.   JOURNAL OF VIROLOGY

Downregulation of Integrin Receptor-Signaling Genes by Epstein-Barr Virus EBNA 3C via Promoter-Proximal and -Distal Binding Elements

(2012) M. J. McClellan et al.   JOURNAL OF VIROLOGY

Direct Recruitment of Polycomb Repressive Complex 1 to Chromatin by Core Binding Transcription Factors

(2012) Ming Yu et al.   MOLECULAR CELL

An integrated encyclopedia of DNA elements in the human genome

(2012)   NATURE

Identifying ChIP-seq enrichment using MACS

(2012) Jianxing Feng et al.   Nature Protocols

BIM promoter directly targeted by EBNA3C in polycomb-mediated repression by EBV

(2012) Kostas Paschos et al.   NUCLEIC ACIDS RESEARCH

Single-vector inducible lentiviral RNAi system for oncology target validation

(2011) Dmitri Wiederschain et al.   CELL CYCLE

Regulation of chromatin by histone modifications

(2011) Andrew J Bannister et al.   CELL RESEARCH

Epstein-Barr virus exploits intrinsic B-lymphocyte transcription programs to achieve immortal cell growth

(2011) B. Zhao et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Epstein-Barr virus nuclear antigens 3C and 3A maintain lymphoblastoid cell growth by repressing p16INK4Aand p14ARFexpression

(2011) Seiji Maruo et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Reciprocal intronic and exonic histone modification regions in humans

(2010) Jason T Huff et al.   NATURE STRUCTURAL & MOLECULAR BIOLOGY

Extensive Co-Operation between the Epstein-Barr Virus EBNA3 Proteins in the Manipulation of Host Gene Expression and Epigenetic Chromatin Modification

(2010) Robert E. White et al.   PLoS One

Epstein-Barr virus nuclear antigen 3C regulated genes in lymphoblastoid cell lines

(2010) B. Zhao et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Epigenetic Repression of p16INK4A by Latent Epstein-Barr Virus Requires the Interaction of EBNA3A and EBNA3C with CtBP

(2010) Lenka Skalska et al.   PLoS Pathogens

Fast and accurate short read alignment with Burrows-Wheeler transform

(2009) H. Li et al.   BIOINFORMATICS

Runx1 promotes B-cell survival and lymphoma development

(2009) Karen Blyth et al.   BLOOD CELLS MOLECULES AND DISEASES

RUNX3-mediated repression of RUNX1 in B cells

(2009) Gareth Brady et al.   JOURNAL OF CELLULAR PHYSIOLOGY

Downregulation of RUNX1 by RUNX3 Requires the RUNX3 VWRPY Sequence and Is Essential for Epstein-Barr Virus-Driven B-Cell Proliferation

(2009) G. Brady et al.   JOURNAL OF VIROLOGY

Differential chromatin marking of introns and expressed exons by H3K36me3

(2009) Paulina Kolasinska-Zwierz et al.   NATURE GENETICS

Recommended tests for association in 2×2 tables

(2009) Stian Lydersen et al.   STATISTICS IN MEDICINE

Differential Gene Expression Patterns of EBV Infected EBNA-3A Positive and Negative Human B Lymphocytes

(2009) Marie L. Hertle et al.   PLoS Pathogens

Model-based Analysis of ChIP-Seq (MACS)

(2008) Yong Zhang et al.   GENOME BIOLOGY