Anti-Inflammatory Effects of Ginsenoside-Rh2 Inhibits LPS-Induced Activation of Microglia and Overproduction of Inflammatory Mediators Via Modulation of TGF-β1/Smad Pathway
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Title
Anti-Inflammatory Effects of Ginsenoside-Rh2 Inhibits LPS-Induced Activation of Microglia and Overproduction of Inflammatory Mediators Via Modulation of TGF-β1/Smad Pathway
Authors
Keywords
GRh2, Lipopolysaccharide, BV-2 microglia cells, TGF-β1/Smad
Journal
NEUROCHEMICAL RESEARCH
Volume 41, Issue 5, Pages 951-957
Publisher
Springer Nature
Online
2016-01-06
DOI
10.1007/s11064-015-1804-x
References
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- Transforming growth factor-β stimulates β amyloid uptake by microglia through Smad3-dependent mechanisms
- (2012) Juan E. Tichauer et al. JOURNAL OF NEUROSCIENCE RESEARCH
- TGF-β1 Pathway as a New Target for Neuroprotection in Alzheimer's Disease
- (2009) Filippo Caraci et al. CNS Neuroscience & Therapeutics
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- Role of pituitary adenylate cyclase-activating polypeptide (PACAP) in the action of ginsenoside Rh2 against beta-amyloid-induced inhibition of rat brain astrocytes
- (2007) Po-Chuen Shieh et al. NEUROSCIENCE LETTERS
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