Transforming growth factor-β stimulates β amyloid uptake by microglia through Smad3-dependent mechanisms

Proinflammatory Stimuli are Needed for Induction of Microglial Cell-Mediated AβPP244–C and Aβ-Neurotoxicity in Hippocampal Cultures

(2017) Gigliola Ramírez et al.   JOURNAL OF ALZHEIMERS DISEASE

Scavenger receptor class A ligands induce secretion of IL1β and exert a modulatory effect on the inflammatory activation of astrocytes in culture

(2012) B. Godoy et al.   JOURNAL OF NEUROIMMUNOLOGY

Aβ Potentiates Inflammatory Activation of Glial Cells Induced by Scavenger Receptor Ligands and Inflammatory Mediators in Culture

(2012) P. Murgas et al.   NEUROTOXICITY RESEARCH

Physiology of Microglia

(2011) Helmut Kettenmann et al.   PHYSIOLOGICAL REVIEWS

Formation and maintenance of Alzheimer's disease β-amyloid plaques in the absence of microglia

(2009) Stefan A Grathwohl et al.   NATURE NEUROSCIENCE

Tau hyperphosphorylation affects Smad 2/3 translocation

(2009) S. Baig et al.   NEUROSCIENCE

Phospho-control of TGF-β superfamily signaling

(2008) Katharine H Wrighton et al.   CELL RESEARCH

Non-Smad pathways in TGF-β signaling

(2008) Ying E Zhang   CELL RESEARCH

TGFβ-mediated BIM expression and apoptosis are regulated through SMAD3-dependent expression of the MAPK phosphatase MKP2

(2008) Sneha Ramesh et al.   EMBO REPORTS

Reduction of β-amyloid-induced neurotoxicity on hippocampal cell cultures by moderate acidosis is mediated by transforming growth factor β

(2008) R. Uribe-San Martín et al.   NEUROSCIENCE