CLEC5A is critical for dengue virus-induced osteoclast activation and bone homeostasis
Published 2016 View Full Article
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Title
CLEC5A is critical for dengue virus-induced osteoclast activation and bone homeostasis
Authors
Keywords
Osteoclasts, Dengue virus, Syk-coupled myeloid C-type lectin member 5A (CLEC5A)
Journal
JOURNAL OF MOLECULAR MEDICINE-JMM
Volume 94, Issue 9, Pages 1025-1037
Publisher
Springer Nature
Online
2016-03-31
DOI
10.1007/s00109-016-1409-0
References
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- Bisphosphonates: Molecular Mechanisms of Action and Effects on Bone Cells, Monocytes and Macrophages
- (2010) A. J. Roelofs et al. CURRENT PHARMACEUTICAL DESIGN
- Myeloid DAP12-associating lectin (MDL)-1 regulates synovial inflammation and bone erosion associated with autoimmune arthritis
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- (2009) M. Inui et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- WNT/β-Catenin Signaling Is Involved in Regulation of Osteoclast Differentiation by Human Immunodeficiency Virus Protease Inhibitor Ritonavir
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- Enhancement by Tumor Necrosis Factor Alpha of Dengue Virus-Induced Endothelial Cell Production of Reactive Nitrogen and Oxygen Species Is Key to Hemorrhage Development
- (2008) Y.-T. Yen et al. JOURNAL OF VIROLOGY
- CLEC5A is critical for dengue-virus-induced lethal disease
- (2008) Szu-Ting Chen et al. NATURE
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