Hippo Pathway Effector Tead1 Induces Cardiac Fibroblast to Cardiomyocyte Reprogramming
Published 2021 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Hippo Pathway Effector Tead1 Induces Cardiac Fibroblast to Cardiomyocyte Reprogramming
Authors
Keywords
-
Journal
Journal of the American Heart Association
Volume 10, Issue 24, Pages -
Publisher
Ovid Technologies (Wolters Kluwer Health)
Online
2021-12-10
DOI
10.1161/jaha.121.022659
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- TEAD1 protects against necroptosis in postmitotic cardiomyocytes through regulation of nuclear DNA-encoded mitochondrial genes
- (2021) Jinhua Liu et al. CELL DEATH AND DIFFERENTIATION
- The histone reader PHF7 cooperates with the SWI/SNF complex at cardiac super enhancers to promote direct reprogramming
- (2021) Glynnis A. Garry et al. NATURE CELL BIOLOGY
- Tead1 is essential for mitochondrial function in cardiomyocytes
- (2020) Ruya Liu et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Enhanced Generation of Induced Cardiomyocytes Using a Small‐Molecule Cocktail to Overcome Barriers to Cardiac Cellular Reprogramming
- (2020) Vivek P. Singh et al. Journal of the American Heart Association
- Down-regulation of Beclin1 promotes direct cardiac reprogramming
- (2020) Li Wang et al. Science Translational Medicine
- Soft Matrix Promotes Cardiac Reprogramming via Inhibition of YAP/TAZ and Suppression of Fibroblast Signatures
- (2020) Shota Kurotsu et al. Stem Cell Reports
- Tead1 is required for perinatal cardiomyocyte proliferation
- (2019) Ruya Liu et al. PLoS One
- A reference map of murine cardiac transcription factor chromatin occupancy identifies dynamic and conserved enhancers
- (2019) Brynn N. Akerberg et al. Nature Communications
- Stoichiometric optimization of Gata4, Hand2, Mef2c, and Tbx5 expression for contractile cardiomyocyte reprogramming
- (2019) Zhentao Zhang et al. Scientific Reports
- Direct In Vivo Reprogramming with Sendai Virus Vectors Improves Cardiac Function after Myocardial Infarction
- (2018) Kazutaka Miyamoto et al. Cell Stem Cell
- p63 Silencing induces reprogramming of cardiac fibroblasts into cardiomyocyte-like cells
- (2018) Vivekkumar Patel et al. JOURNAL OF THORACIC AND CARDIOVASCULAR SURGERY
- YAP1-TEAD1 signaling controls angiogenesis and mitochondrial biogenesis through PGC1α
- (2018) Akiko Mammoto et al. MICROVASCULAR RESEARCH
- In situ reprogramming to transdifferentiate fibroblasts into cardiomyocytes using adenoviral vectors: Implications for clinical myocardial regeneration
- (2017) Megumi Mathison et al. JOURNAL OF THORACIC AND CARDIOVASCULAR SURGERY
- Cardiac reprogramming factor Gata4 reduces postinfarct cardiac fibrosis through direct repression of the profibrotic mediator snail
- (2017) Megumi Mathison et al. JOURNAL OF THORACIC AND CARDIOVASCULAR SURGERY
- Regulation of the Hippo Pathway Transcription Factor TEAD
- (2017) Kimberly C. Lin et al. TRENDS IN BIOCHEMICAL SCIENCES
- Core Transcription Factors, MicroRNAs and Small Molecules Drive Transdifferentiation of Human Fibroblasts Towards The Cardiac Cell Lineage
- (2017) Nicolas Christoforou et al. Scientific Reports
- Notch Inhibition Enhances Cardiac Reprogramming by Increasing MEF2C Transcriptional Activity
- (2017) Maria Abad et al. Stem Cell Reports
- Bmi1 Is a Key Epigenetic Barrier to Direct Cardiac Reprogramming
- (2016) Yang Zhou et al. Cell Stem Cell
- Chemical Enhancement of In Vitro and In Vivo Direct Cardiac ReprogrammingClinical Perspective
- (2016) Tamer M. A. Mohamed et al. CIRCULATION
- The Biological Basis for Cardiac Repair After Myocardial Infarction
- (2016) Sumanth D. Prabhu et al. CIRCULATION RESEARCH
- Mechanisms of Hippo pathway regulation
- (2016) Zhipeng Meng et al. GENES & DEVELOPMENT
- Generation of an inducible fibroblast cell line for studying direct cardiac reprogramming
- (2016) Haley Ruth Vaseghi et al. GENESIS
- MiR‐590 Promotes Transdifferentiation of Porcine and Human Fibroblasts Toward a Cardiomyocyte‐Like Fate by Directly Repressing Specificity Protein 1
- (2016) Vivek P. Singh et al. Journal of the American Heart Association
- Hippo Pathway in Organ Size Control, Tissue Homeostasis, and Cancer
- (2015) Fa-Xing Yu et al. CELL
- The Hippo Pathway in Heart Development, Regeneration, and Diseases
- (2015) Q. Zhou et al. CIRCULATION RESEARCH
- Akt1/protein kinase B enhances transcriptional reprogramming of fibroblasts to functional cardiomyocytes
- (2015) Huanyu Zhou et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Disease implications of the Hippo/YAP pathway
- (2015) Steven W. Plouffe et al. TRENDS IN MOLECULAR MEDICINE
- High-efficiency reprogramming of fibroblasts into cardiomyocytes requires suppression of pro-fibrotic signalling
- (2015) Yuanbiao Zhao et al. Nature Communications
- Direct Reprogramming of Human Fibroblasts to Functional and Expandable Hepatocytes
- (2014) Pengyu Huang et al. Cell Stem Cell
- “Triplet” polycistronic vectors encoding Gata4, Mef2c, and Tbx5 enhances postinfarct ventricular functional improvement compared with singlet vectors
- (2014) Megumi Mathison et al. JOURNAL OF THORACIC AND CARDIOVASCULAR SURGERY
- Epidemiology of Heart Failure and Scope of the Problem
- (2013) Longjian Liu et al. CARDIOLOGY CLINICS
- Optimization of direct fibroblast reprogramming to cardiomyocytes using calcium activity as a functional measure of success
- (2013) Russell C. Addis et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Reprogramming of human fibroblasts toward a cardiac fate
- (2013) Y.-J. Nam et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Hippo pathway effector Yap promotes cardiac regeneration
- (2013) Mei Xin et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- In Vivo Cardiac Cellular Reprogramming Efficacy Is Enhanced by Angiogenic Preconditioning of the Infarcted Myocardium With Vascular Endothelial Growth Factor
- (2013) Megumi Mathison et al. Journal of the American Heart Association
- Direct Reprogramming of Human Fibroblasts toward a Cardiomyocyte-like State
- (2013) Ji-Dong Fu et al. Stem Cell Reports
- MicroRNA-Mediated In Vitro and In Vivo Direct Reprogramming of Cardiac Fibroblasts to Cardiomyocytes
- (2012) Tilanthi M. Jayawardena et al. CIRCULATION RESEARCH
- Heart repair by reprogramming non-myocytes with cardiac transcription factors
- (2012) Kunhua Song et al. NATURE
- In vivo reprogramming of murine cardiac fibroblasts into induced cardiomyocytes
- (2012) Li Qian et al. NATURE
- Co-occupancy by multiple cardiac transcription factors identifies transcriptional enhancers active in heart
- (2011) A. He et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Hippo Pathway Inhibits Wnt Signaling to Restrain Cardiomyocyte Proliferation and Heart Size
- (2011) T. Heallen et al. SCIENCE
- The Cardiac Transcription Network Modulated by Gata4, Mef2a, Nkx2.5, Srf, Histone Modifications, and MicroRNAs
- (2011) Jenny Schlesinger et al. PLoS Genetics
- Direct Reprogramming of Fibroblasts into Functional Cardiomyocytes by Defined Factors
- (2010) Masaki Ieda et al. CELL
- Mammalian Tead proteins regulate cell proliferation and contact inhibition as transcriptional mediators of Hippo signaling
- (2008) M. Ota et al. DEVELOPMENT
- MCAT Elements and the TEF-1 Family of Transcription Factors in Muscle Development and Disease
- (2007) Tadashi Yoshida ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
- Stimulation of mitochondrial biogenesis and autophagy by lipopolysaccharide in the neonatal rat cardiomyocyte protects against programmed cell death
- (2007) Diane L.M. Hickson-Bick et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExplorePublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More