Excessive Hypoxia‐Inducible Factor‐1α Expression Induces Cardiac Rupture via p53‐Dependent Apoptosis After Myocardial Infarction
Published 2021 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Excessive Hypoxia‐Inducible Factor‐1α Expression Induces Cardiac Rupture via p53‐Dependent Apoptosis After Myocardial Infarction
Authors
Keywords
-
Journal
Journal of the American Heart Association
Volume 10, Issue 17, Pages -
Publisher
Ovid Technologies (Wolters Kluwer Health)
Online
2021-09-02
DOI
10.1161/jaha.121.020895
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Heart Rate Reduction with Ivabradine Prevents Cardiac Rupture after Myocardial Infarction in Mice
- (2021) Masataka Ikeda et al. CARDIOVASCULAR DRUGS AND THERAPY
- Roxadustat Markedly Reduces Myocardial Ischemia Reperfusion Injury in Mice
- (2020) Hiroko Deguchi et al. CIRCULATION JOURNAL
- Reduction in ST-Segment Elevation Cardiac Catheterization Laboratory Activations in the United States During COVID-19 Pandemic
- (2020) Santiago Garcia et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- COVID-19 pandemic and admission rates for and management of acute coronary syndromes in England
- (2020) Marion M Mafham et al. LANCET
- Mitochondria-dependent ferroptosis plays a pivotal role in doxorubicin cardiotoxicity
- (2020) Tomonori Tadokoro et al. JCI Insight
- HIF-1-Dependent Reprogramming of Glucose Metabolic Pathway of Cancer Cells and Its Therapeutic Significance
- (2019) Ayako Nagao et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
- Caspases in Cell Death, Inflammation, and Disease
- (2019) Nina Van Opdenbosch et al. IMMUNITY
- Temporal Trends and Outcomes of Mechanical Complications in Patients With Acute Myocardial Infarction
- (2019) Ayman Elbadawi et al. JACC-Cardiovascular Interventions
- Mechano-chronotropic Unloading During the Acute Phase of Myocardial Infarction Markedly Reduces Infarct Size via the Suppression of Myocardial Oxygen Consumption
- (2018) Genya Sunagawa et al. Journal of Cardiovascular Translational Research
- Cardiomyocyte gene programs encoding morphological and functional signatures in cardiac hypertrophy and failure
- (2018) Seitaro Nomura et al. Nature Communications
- Beta-blockers reduced the risk of cardiac rupture in patients with acute myocardial infarction: A meta-analysis of randomized control trials
- (2017) Wei Gong et al. INTERNATIONAL JOURNAL OF CARDIOLOGY
- Twinkle overexpression prevents cardiac rupture after myocardial infarction by alleviating impaired mitochondrial biogenesis
- (2016) Takahiro Inoue et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Regulation of the p53 response and its relationship to cancer
- (2015) David W. Meek BIOCHEMICAL JOURNAL
- HIF Hydroxylase Pathways in Cardiovascular Physiology and Medicine
- (2015) T. Bishop et al. CIRCULATION RESEARCH
- Coronary microvascular obstruction in acute myocardial infarction
- (2015) Giampaolo Niccoli et al. EUROPEAN HEART JOURNAL
- Impact of Ivabradine on Central Aortic Blood Pressure and Myocardial Perfusion in Patients With Stable Coronary Artery Disease
- (2015) Jean-Guillaume Dillinger et al. HYPERTENSION
- Depletion of PHD3 protects heart from ischemia/reperfusion injury by inhibiting cardiomyocyte apoptosis
- (2015) Liang Xie et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- The Akt-mTOR axis is a pivotal regulator of eccentric hypertrophy during volume overload
- (2015) Masataka Ikeda et al. Scientific Reports
- Overexpression of TFAM or Twinkle Increases mtDNA Copy Number and Facilitates Cardioprotection Associated with Limited Mitochondrial Oxidative Stress
- (2015) Masataka Ikeda et al. PLoS One
- HIF Transcription Factors, Inflammation, and Immunity
- (2014) Asis Palazon et al. IMMUNITY
- Nontranscriptional Role of Hif-1α in Activation of γ-Secretase and Notch Signaling in Breast Cancer
- (2014) Jennifer C. Villa et al. Cell Reports
- HIF-1 and paradoxical phenomena in cardioprotection: EXPERT'S PERSPECTIVE
- (2012) G. Heusch CARDIOVASCULAR RESEARCH
- Unfavourable consequences of chronic cardiac HIF-1α stabilization
- (2012) Marion Hölscher et al. CARDIOVASCULAR RESEARCH
- Hypoxia-Inducible Factors in Physiology and Medicine
- (2012) Gregg L. Semenza CELL
- Recombinant mitochondrial transcription factor A protein inhibits nuclear factor of activated T cells signaling and attenuates pathological hypertrophy of cardiac myocytes
- (2012) Takeo Fujino et al. MITOCHONDRION
- Cardiomyocyte-specific Prolyl-4-hydroxylase Domain 2 Knock Out Protects from Acute Myocardial Ischemic Injury
- (2011) Marion Hölscher et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Disruption of Hypoxia-Inducible Transcription Factor-Prolyl Hydroxylase Domain-1 (PHD-1−/−) Attenuates Ex Vivo Myocardial Ischemia/Reperfusion Injury Through Hypoxia-Inducible Factor-1α Transcription Factor and Its Target Genes in Mice
- (2010) Ram Sudheer Adluri et al. ANTIOXIDANTS & REDOX SIGNALING
- Loss of Hypoxia-Inducible Factor Prolyl Hydroxylase Activity in Cardiomyocytes Phenocopies Ischemic Cardiomyopathy
- (2010) Javid Moslehi et al. CIRCULATION
- Association Between Timeliness of Reperfusion Therapy and Clinical Outcomes in ST-Elevation Myocardial Infarction
- (2010) Laurie Lambert JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION
- Hypoxia-Inducible Factors and the Response to Hypoxic Stress
- (2010) Amar J. Majmundar et al. MOLECULAR CELL
- Conditional HIF-1α Expression Produces a Reversible Cardiomyopathy
- (2010) Raffi Bekeredjian et al. PLoS One
- Cardiac Rupture After Myocardial Infarction
- (2009) David C. Sane et al. Cardiology in Review
- Changes in Hospital Mortality Rates in 425 Patients With Acute ST-Elevation Myocardial Infarction and Cardiac Rupture Over a 30-Year Period
- (2008) Jaume Figueras et al. CIRCULATION
- Oxygen Sensing by Metazoans: The Central Role of the HIF Hydroxylase Pathway
- (2008) William G. Kaelin et al. MOLECULAR CELL
- Somatic inactivation of the PHD2 prolyl hydroxylase causes polycythemia and congestive heart failure
- (2007) Y. A. Minamishima et al. BLOOD
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationFind the ideal target journal for your manuscript
Explore over 38,000 international journals covering a vast array of academic fields.
Search