MAX mutant small-cell lung cancers exhibit impaired activities of MGA-dependent noncanonical polycomb repressive complex
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Title
MAX mutant small-cell lung cancers exhibit impaired activities of MGA-dependent noncanonical polycomb repressive complex
Authors
Keywords
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Journal
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
Volume 118, Issue 37, Pages e2024824118
Publisher
Proceedings of the National Academy of Sciences
Online
2021-09-15
DOI
10.1073/pnas.2024824118
References
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Note: Only part of the references are listed.- MAX Functions as a Tumor Suppressor and Rewires Metabolism in Small Cell Lung Cancer
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- Multi-Omics Analysis Identifies MGA as a Negative Regulator of the MYC Pathway in Lung Adenocarcinoma
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- MGA, L3MBTL2 and E2F6 determine genomic binding of the non-canonical Polycomb repressive complex PRC1.6
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- The MYC transcription factor network: balancing metabolism, proliferation and oncogenesis
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- PCGF6-PRC1 suppresses premature differentiation of mouse embryonic stem cells by regulating germ cell-related genes
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- Loss of MAX results in meiotic entry in mouse embryonic and germline stem cells
- (2016) Ayumu Suzuki et al. Nature Communications
- A High-Density Map for Navigating the Human Polycomb Complexome
- (2016) Simon Hauri et al. Cell Reports
- ASCL1 and NEUROD1 Reveal Heterogeneity in Pulmonary Neuroendocrine Tumors and Regulate Distinct Genetic Programs
- (2016) Mark D. Borromeo et al. Cell Reports
- Functional interactions among members of the MAX and MLX transcriptional network during oncogenesis
- (2015) Daniel Diolaiti et al. Biochimica et Biophysica Acta-Gene Regulatory Mechanisms
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- Exome sequencing identifies somatic mutations of DDX3X in natural killer/T-cell lymphoma
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- MAX Inactivation in Small Cell Lung Cancer Disrupts MYC-SWI/SNF Programs and Is Synthetic Lethal with BRG1
- (2013) O. A. Romero et al. Cancer Discovery
- The Polycomb Group Protein L3mbtl2 Assembles an Atypical PRC1-Family Complex that Is Essential in Pluripotent Stem Cells and Early Development
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