miR-135a Reduces Osteosarcoma Pulmonary Metastasis by Targeting Both BMI1 and KLF4
Published 2021 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
miR-135a Reduces Osteosarcoma Pulmonary Metastasis by Targeting Both BMI1 and KLF4
Authors
Keywords
-
Journal
Frontiers in Oncology
Volume 11, Issue -, Pages -
Publisher
Frontiers Media SA
Online
2021-03-22
DOI
10.3389/fonc.2021.620295
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Regulation of Krüppel-Like Factor 4 (KLF4) expression through the transcription factor Yin-Yang 1 (YY1) in non-Hodgkin B-cell lymphoma
- (2019) Mario Morales-Martinez et al. Oncotarget
- miR-135 regulated breast cancer proliferation and epithelial-mesenchymal transition acts by the Wnt/β-catenin signaling pathway
- (2019) Daqiong Jiang et al. INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE
- miR‐134 inhibits osteosarcoma cell invasion and metastasis through targeting MMP 1 and MMP 3 in vitro and in vivo
- (2019) Cheng‐long Chen et al. FEBS LETTERS
- CUL4B regulates cancer stem‐like traits of prostate cancer cells by targeting BMI1 via miR200b/c
- (2019) Meng Jiao et al. PROSTATE
- Advances in immune checkpoint inhibitors for bone sarcoma therapy
- (2019) Pichaya Thanindratarn et al. Journal of Bone Oncology
- LncRNA DANCR promotes the proliferation, migration, and invasion of tongue squamous cell carcinoma cells through miR-135a-5p/KLF8 axis
- (2019) Ying Zheng et al. Cancer Cell International
- CircPLEKHM3 acts as a tumor suppressor through regulation of the miR-9/BRCA1/DNAJB6/KLF4/AKT1 axis in ovarian cancer
- (2019) Lei Zhang et al. Molecular Cancer
- Silencing lncRNA ZFAS1 or elevated microRNA-135a represses proliferation, migration, invasion and resistance to apoptosis of osteosarcoma cells
- (2019) Zilong Zhao et al. Cancer Cell International
- KLF4 functions as an oncogene in promoting cancer stem cell-like characteristics in osteosarcoma cells
- (2018) Xiao-tian Qi et al. ACTA PHARMACOLOGICA SINICA
- Cancer statistics, 2018
- (2018) Rebecca L. Siegel et al. CA-A CANCER JOURNAL FOR CLINICIANS
- MicroRNA-134 inhibits osteosarcoma angiogenesis and proliferation by targeting the VEGFA/VEGFR1 pathway
- (2018) Long Zhang et al. FEBS Journal
- Inhibition of BMI1, a therapeutic approach in endometrial cancer.
- (2018) Megan Buechel et al. MOLECULAR CANCER THERAPEUTICS
- Krüppel-like factor 4 (KLF4): What we currently know
- (2017) Amr M. Ghaleb et al. GENE
- An Image-Based miRNA Screen Identifies miRNA-135s As Regulators of CNS Axon Growth and Regeneration by Targeting Krüppel-like Factor 4
- (2017) Eljo Y. van Battum et al. JOURNAL OF NEUROSCIENCE
- Bmi-1-targeting suppresses osteosarcoma aggressiveness through the NF-κB signaling pathway
- (2017) Jiaguo Liu et al. Molecular Medicine Reports
- Inhibition of KLF4 by Statins Reverses Adriamycin-Induced Metastasis and Cancer Stemness in Osteosarcoma Cells
- (2017) Yangling Li et al. Stem Cell Reports
- Inhibition of BMI1 induces autophagy-mediated necroptosis
- (2016) Anindya Dey et al. Autophagy
- miRNA-135a promotes hepatocellular carcinoma cell migration and invasion by targeting forkhead box O1
- (2016) Yue-Bin Zeng et al. Cancer Cell International
- hsa-miR-135a-1 inhibits prostate cancer cell growth and migration by targeting EGFR
- (2016) Bin Xu et al. TUMOR BIOLOGY
- Krüppel-like factor 4 promotes human osteosarcoma growth and metastasis via regulating CRYAB expression
- (2016) Lu Zhang et al. Oncotarget
- TWIST1 and BMI1 in Cancer Metastasis and Chemoresistance
- (2016) Hong Ren et al. Journal of Cancer
- Sialyltransferase7A, a Klf4-responsive gene, promotes cardiomyocyte apoptosis during myocardial infarction
- (2015) Dongmei Zhang et al. BASIC RESEARCH IN CARDIOLOGY
- Targeting of Runx2 by miR-135 and miR-203 Impairs Progression of Breast Cancer and Metastatic Bone Disease
- (2015) H. Taipaleenmaki et al. CANCER RESEARCH
- Combination immunotherapy with α-CTLA-4 and α-PD-L1 antibody blockade prevents immune escape and leads to complete control of metastatic osteosarcoma
- (2015) Danielle M Lussier et al. Journal for ImmunoTherapy of Cancer
- MiR-138 and MiR-135 Directly Target Focal Adhesion Kinase, Inhibit Cell Invasion, and Increase Sensitivity to Chemotherapy in Cancer Cells
- (2014) Vita Golubovskaya et al. Anti-Cancer Agents in Medicinal Chemistry
- The Tumor-Suppressive MicroRNA-135b Targets c-Myc in Osteoscarcoma
- (2014) Zheng Liu et al. PLoS One
- Overexpression of Bmi-1 contributes to the invasion and metastasis of hepatocellular carcinoma by increasing the expression of matrix metalloproteinase (MMP)-2, MMP-9 and vascular endothelial growth factor via the PTEN/PI3K/Akt pathway
- (2013) XIAOLEI LI et al. INTERNATIONAL JOURNAL OF ONCOLOGY
- Tumor and its microenvironment: A synergistic interplay
- (2013) Veronica Catalano et al. SEMINARS IN CANCER BIOLOGY
- Bmi-1 promotes the aggressiveness of glioma via activating the NF-kappaB/MMP-9 signaling pathway
- (2012) Lili Jiang et al. BMC CANCER
- The extracellular matrix: A dynamic niche in cancer progression
- (2012) Pengfei Lu et al. JOURNAL OF CELL BIOLOGY
- Role of BMI1, a Stem Cell Factor, in Cancer Recurrence and Chemoresistance: Preclinical and Clinical Evidences
- (2012) Hifzur Rahman Siddique et al. STEM CELLS
- BMI1 as a novel target for drug discovery in cancer
- (2011) Liangxian Cao et al. JOURNAL OF CELLULAR BIOCHEMISTRY
- Overexpression of BMI-1 Promotes Cell Growth and Resistance to Cisplatin Treatment in Osteosarcoma
- (2011) Zhihong Wu et al. PLoS One
- Pleiotropic roles of matrix metalloproteinases in tumor angiogenesis: Contrasting, overlapping and compensatory functions
- (2009) Elena I. Deryugina et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH
- The extracellular matrix in development and morphogenesis: A dynamic view
- (2009) Tania Rozario et al. DEVELOPMENTAL BIOLOGY
Find the ideal target journal for your manuscript
Explore over 38,000 international journals covering a vast array of academic fields.
SearchAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started