Activating transcription factor 3 involved in Pseudomonas aeruginosa PAO1-induced macrophage senescence

Cellular senescence as a potential mediator of COVID‐19 severity in the elderly

(2020) Jamil Nehme et al.   AGING CELL

Function, Oxidative, and Inflammatory Stress Parameters in Immune Cells as Predictive Markers of Lifespan throughout Aging

(2019) Irene Martínez de Toda et al.   Oxidative Medicine and Cellular Longevity

Using ROS as a Second Messenger, NADPH Oxidase 2 Mediates Macrophage Senescence via Interaction with NF-κB during Pseudomonas aeruginosa Infection

(2018) Hui Li et al.   Oxidative Medicine and Cellular Longevity

Early life infection and host senescence

(2018) Emmanuel Guivier et al.   EXPERIMENTAL GERONTOLOGY

Macrophage de novo NAD+ synthesis specifies immune function in aging and inflammation

(2018) Paras S. Minhas et al.   NATURE IMMUNOLOGY

Activating transcription factor 3 protects mice against pseudomonas aeruginosa-induced acute lung injury by interacting with lipopolysaccharide binding protein

(2017) Yunfeng Zhao et al.   MOLECULAR IMMUNOLOGY

Aging of mice is associated with p16(Ink4a)- and β-galactosidase-positive macrophage accumulation that can be induced in young mice by senescent cells

(2016) Brandon M. Hall et al.   Aging-US

Sepsis: pathophysiology and clinical management

(2016) Jeffrey E Gotts et al.   BMJ-British Medical Journal

Sepsis: pathophysiology and clinical management

(2016) Jeffrey E Gotts et al.   BMJ-British Medical Journal

Pseudomonas aeruginosa N-3-oxo-dodecanoyl-homoserine Lactone Elicits Changes in Cell Volume, Morphology, and AQP9 Characteristics in Macrophages

(2016) Angelika Holm et al.   Frontiers in Cellular and Infection Microbiology

Acrylamide Induces Senescence in Macrophages through a Process Involving ATF3, ROS, p38/JNK, and a Telomerase-Independent Pathway

(2015) Kyung-Ho Kim et al.   CHEMICAL RESEARCH IN TOXICOLOGY

Initial Host Response to Bacteria in the Murine Lung Differs Between Pseudomonas aeruginosa, Staphylococcus aureus and Streptococcus pneumoniae

(2015) Liselotte Preu et al.   INFLAMMATION

Activating transcription factor 3 represses inflammatory responses by binding to the p65 subunit of NF-κB

(2015) Ji-Woong Kwon et al.   Scientific Reports

Density-gradient centrifugation enables the purification of cultured corneal endothelial cells for cell therapy by eliminating senescent cells

(2015) Naoki Okumura et al.   Scientific Reports

ATF3-Mediated NRF2/HO-1 Signaling Regulates TLR4 Innate Immune Responses in Mouse Liver Ischemia/Reperfusion Injury

(2014) J. Rao et al.   AMERICAN JOURNAL OF TRANSPLANTATION

ATF3 attenuates cyclosporin A-induced nephrotoxicity by downregulating CHOP in HK-2 cells

(2014) Yong-Min Choi et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS

Resveratrol increases anti-aging Klotho gene expression via the activating transcription factor 3/c-Jun complex-mediated signaling pathway

(2014) Shih-Che Hsu et al.   INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY

The Activating Transcription Factor 3 Protein Suppresses the Oncogenic Function of Mutant p53 Proteins

(2014) Saisai Wei et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

ATF3 Confers Resistance to Pneumococcal Infection Through Positive Regulation of Cytokine Production

(2014) Cuong Thach Nguyen et al.   JOURNAL OF INFECTIOUS DISEASES

Apoptosis are induced in J774 macrophages upon phagocytosis and killing of Pseudomonas aeruginosa

(2013) Jianling Zhang et al.   CELLULAR IMMUNOLOGY

NF-κB Inhibition after Cecal Ligation and Puncture Reduces Sepsis-Associated Lung Injury without Altering Bacterial Host Defense

(2013) Hui Li et al.   MEDIATORS OF INFLAMMATION

ROS-induced ATF3 causes susceptibility to secondary infections during sepsis-associated immunosuppression

(2011) Wolfram Hoetzenecker et al.   NATURE MEDICINE

Opposing roles for calcineurin and ATF3 in squamous skin cancer

(2010) Xunwei Wu et al.   NATURE

Function of C/EBPδ in a regulatory circuit that discriminates between transient and persistent TLR4-induced signals

(2009) Vladimir Litvak et al.   NATURE IMMUNOLOGY