Pathogen-Associated Molecules from Gut Translocation Enhance Severity of Cecal Ligation and Puncture Sepsis in Iron-Overload β-Thalassemia Mice

Leaky-gut enhanced lupus progression in the Fc gamma receptor-IIb deficient and pristane-induced mouse models of lupus

(2020) Arthid Thim-uam et al.   Scientific Reports

Gut leakage enhances sepsis susceptibility in iron-overloaded β-thalassemia mice through macrophage hyperinflammatory responses

(2020) Peerapat Visitchanakun et al.   AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY

Syk inhibitor attenuates inflammation in lupus mice from FcgRIIb deficiency but not in pristane induction: the influence of lupus pathogenesis on the therapeutic effect

(2020) Jiraphorn Issara-Amphorn et al.   LUPUS

Over-expression of miR-223 induces M2 macrophage through glycolysis alteration and attenuates LPS-induced sepsis mouse model, the cell-based therapy in sepsis

(2020) Cong Phi Dang et al.   PLoS One

Defective Neutrophil Function in Patients with Sepsis Is Mostly Restored by ex vivo Ascorbate Incubation

(2020) Kritsanawan Sae-khow et al.   Journal of Inflammation Research

Decreased Protein Kinase C-β Type II Associated with the Prominent Endotoxin Exhaustion in the Macrophage of FcGRIIb−/− Lupus Prone Mice is Revealed by Phosphoproteomic Analysis

(2019) Thunnicha Ondee et al.   INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES

Gut Leakage of Fungal-Derived Inflammatory Mediators: Part of a Gut-Liver-Kidney Axis in Bacterial Sepsis

(2019) Panomwat Amornphimoltham et al.   DIGESTIVE DISEASES AND SCIENCES

Blockade of PD-1 Attenuated Post-Sepsis Aspergillosis via The Activation Of IFN-γ and The Dampening of IL-10

(2019) Chau Tran Bao Vu et al.   SHOCK

Effects of intracellular iron overload on cell death and identification of potent cell death inhibitors

(2018) Shenglin Fang et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS

An energetic view of stress: Focus on mitochondria

(2018) Martin Picard et al.   FRONTIERS IN NEUROENDOCRINOLOGY

A combination Prussian blue – hematoxylin and eosin staining technique for identification of iron and other histological features

(2018) Katelyn Rowatt et al.   JOURNAL OF HISTOTECHNOLOGY

The Synergy of Endotoxin and (1→3)-β-D-Glucan, from Gut Translocation, Worsens Sepsis Severity in a Lupus Model of Fc Gamma Receptor IIb-Deficient Mice

(2018) Jiraphorn Issara-Amphorn et al.   Journal of Innate Immunity

Oral administration of liquid iron preparation containing excess iron induces intestine and liver injury, impairs intestinal barrier function and alters the gut microbiota in rats

(2018) Shenglin Fang et al.   JOURNAL OF TRACE ELEMENTS IN MEDICINE AND BIOLOGY

Gastrointestinal Colonization of Candida Albicans Increases Serum (1→3)-β-D-Glucan, without Candidemia, and Worsens Cecal Ligation and Puncture Sepsis in Murine Model

(2018) Wimonrat Panpetch et al.   SHOCK

Iron overloaded polarizes macrophage to proinflammation phenotype through ROS/acetyl-p53 pathway

(2018) Yun Zhou et al.   Cancer Medicine

Intestinal fungi contribute to development of alcoholic liver disease

(2017) An-Ming Yang et al.   JOURNAL OF CLINICAL INVESTIGATION

The role of macrophages in the susceptibility of Fc gamma receptor IIb deficient mice to Cryptococcus neoformans

(2017) Saowapha Surawut et al.   Scientific Reports

Oral administration of live- or heat-killed Candida albicans worsened cecal ligation and puncture sepsis in a murine model possibly due to an increased serum (1→3)-β-D-glucan

(2017) Wimonrat Panpetch et al.   PLoS One

Gastrointestinal Leakage Detected by Serum (1→3)-β-D-Glucan in Mouse Models and a Pilot Study in Patients with Sepsis

(2016) Asada Leelahavanichkul et al.   SHOCK

Influence of pre-existing inflammation on the outcome of acute coronary syndrome: a cross-sectional study

(2016) Jacob Odeberg et al.   BMJ Open

Paradoxical Roles of the Neutrophil in Sepsis: Protective and Deleterious

(2016) Fabiane Sônego et al.   Frontiers in Immunology

High-dose ascorbate with low-dose amphotericin B attenuates severity of disease in a model of the reappearance of candidemia during sepsis in the mouse

(2015) Asada Leelahavanichkul et al.   AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY

Severe bacterial infections in patients with non-transfusion-dependent thalassemia: prevalence and clinical risk factors

(2015) Nattiya Teawtrakul et al.   INTERNATIONAL JOURNAL OF INFECTIOUS DISEASES

Macrophage Polarization in Inflammatory Diseases

(2014) Yan-Cun Liu et al.   International Journal of Biological Sciences

Functional Roles of Syk in Macrophage-Mediated Inflammatory Responses

(2014) Young-Su Yi et al.   MEDIATORS OF INFLAMMATION

Epithelial Cell Shedding and Barrier Function

(2014) J. M. Williams et al.   VETERINARY PATHOLOGY

Murine Models of Candida Gastrointestinal Colonization and Dissemination

(2013) Andrew Y. Koh   EUKARYOTIC CELL

New updating into hemoglobinopathies

(2012) S. Fucharoen et al.   International Journal of Laboratory Hematology

How I treat thalassemia

(2011) E. A. Rachmilewitz et al.   BLOOD

Chronic kidney disease worsens sepsis and sepsis-induced acute kidney injury by releasing High Mobility Group Box Protein-1

(2011) Asada Leelahavanichkul et al.   KIDNEY INTERNATIONAL

Interleukin-33 attenuates sepsis by enhancing neutrophil influx to the site of infection

(2010) Jose C Alves-Filho et al.   NATURE MEDICINE

Ineffective erythropoiesis and thalassemias

(2009) Stefano Rivella   CURRENT OPINION IN HEMATOLOGY

Animal models of sepsis and sepsis-induced kidney injury

(2009) Kent Doi et al.   JOURNAL OF CLINICAL INVESTIGATION

Dectin-1 synergizes with TLR2 and TLR4 for cytokine production in human primary monocytes and macrophages

(2008) Gerben Ferwerda et al.   CELLULAR MICROBIOLOGY

Syk kinase is required for collaborative cytokine production induced through Dectin-1 and Toll-like receptors

(2008) Kevin M. Dennehy et al.   EUROPEAN JOURNAL OF IMMUNOLOGY

Pre-existing renal disease promotes sepsis-induced acute kidney injury and worsens outcome

(2008) Kent Doi et al.   KIDNEY INTERNATIONAL