Therapeutic Mechanism of Macrophage Inflammatory Protein 1 α Neutralizing Antibody (CCL3) in Clostridium difficile Infection in Mice
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Title
Therapeutic Mechanism of Macrophage Inflammatory Protein 1 α Neutralizing Antibody (CCL3) in Clostridium difficile Infection in Mice
Authors
Keywords
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Journal
JOURNAL OF INFECTIOUS DISEASES
Volume -, Issue -, Pages -
Publisher
Oxford University Press (OUP)
Online
2019-12-03
DOI
10.1093/infdis/jiz640
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Note: Only part of the references are listed.- Clostridium difficile toxins A and B decrease intestinal SLC26A3 protein expression
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- Critical Roles of Clostridium difficile Toxin B Enzymatic Activities in Pathogenesis
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- Slc26a3 deficiency is associated with loss of colonic HCO3−secretion, absence of a firm mucus layer and barrier impairment in mice
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- Guidelines for Diagnosis, Treatment and Prevention of Clostridium difficile Infections
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