New Alzheimer’s disease model mouse specialized for analyzing the function and toxicity of intraneuronal Amyloid β oligomers
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Title
New Alzheimer’s disease model mouse specialized for analyzing the function and toxicity of intraneuronal Amyloid β oligomers
Authors
Keywords
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Journal
Scientific Reports
Volume 9, Issue 1, Pages -
Publisher
Springer Science and Business Media LLC
Online
2019-11-22
DOI
10.1038/s41598-019-53415-8
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Note: Only part of the references are listed.- Reduction in open field activity in the absence of memory deficits in the App NL−G−F knock-in mouse model of Alzheimer’s disease
- (2018) Lauren S. Whyte et al. BEHAVIOURAL BRAIN RESEARCH
- APP as a Protective Factor in Acute Neuronal Insults
- (2017) Dimitri Hefter et al. Frontiers in Molecular Neuroscience
- Large Soluble Oligomers of Amyloid β-Protein from Alzheimer Brain Are Far Less Neuroactive Than the Smaller Oligomers to Which They Dissociate
- (2016) Ting Yang et al. JOURNAL OF NEUROSCIENCE
- Amyloid Precursor Protein Protects Neuronal Network Function after Hypoxia via Control of Voltage-Gated Calcium Channels
- (2016) D. Hefter et al. JOURNAL OF NEUROSCIENCE
- Development of new fusion proteins for visualizing amyloid-β oligomers in vivo
- (2016) Tomoyo Ochiishi et al. Scientific Reports
- The Neuroprotective Properties of the Amyloid Precursor Protein Following Traumatic Brain Injury
- (2016) Stephanie Plummer et al. Aging and Disease
- Viral gene transfer of APPsα rescues synaptic failure in an Alzheimer’s disease mouse model
- (2015) Romain Fol et al. ACTA NEUROPATHOLOGICA
- The aqueous phase of Alzheimer's disease brain contains assemblies built from ∼4 and ∼7 kDa Aβ species
- (2015) Jessica M. Mc Donald et al. Alzheimers & Dementia
- Intracellular amyloid β oligomers impair organelle transport and induce dendritic spine loss in primary neurons
- (2015) Tomohiro Umeda et al. Acta Neuropathologica Communications
- Acute function of secreted amyloid precursor protein fragment APPsα in synaptic plasticity
- (2014) Meike Hick et al. ACTA NEUROPATHOLOGICA
- Peripheral Administration of a Humanized Anti-PrP Antibody Blocks Alzheimer's Disease A Synaptotoxicity
- (2014) I. Klyubin et al. JOURNAL OF NEUROSCIENCE
- Single App knock-in mouse models of Alzheimer's disease
- (2014) Takashi Saito et al. NATURE NEUROSCIENCE
- mGlu5 receptors and cellular prion protein mediate amyloid-β-facilitated synaptic long-term depression in vivo
- (2014) Neng-Wei Hu et al. Nature Communications
- Hippocampal Network Oscillations in APP/APLP2-Deficient Mice
- (2013) Xiaomin Zhang et al. PLoS One
- Trafficking and Proteolytic Processing of APP
- (2013) C. Haass et al. Cold Spring Harbor Perspectives in Medicine
- Alzheimer brain-derived amyloid β-protein impairs synaptic remodeling and memory consolidation
- (2012) Gilyana G. Borlikova et al. NEUROBIOLOGY OF AGING
- Transsynaptic Signaling by Activity-Dependent Cleavage of Neuroligin-1
- (2012) Rui T. Peixoto et al. NEURON
- A Subtype-Specific Function for the Extracellular Domain of Neuroligin 1 in Hippocampal LTP
- (2012) Seth L. Shipman et al. NEURON
- Network Oscillations in Rod-Degenerated Mouse Retinas
- (2011) J. Menzler et al. JOURNAL OF NEUROSCIENCE
- Alzheimer's Disease Brain-Derived Amyloid- -Mediated Inhibition of LTP In Vivo Is Prevented by Immunotargeting Cellular Prion Protein
- (2011) A. E. Barry et al. JOURNAL OF NEUROSCIENCE
- Soluble A Oligomers Inhibit Long-Term Potentiation through a Mechanism Involving Excessive Activation of Extrasynaptic NR2B-Containing NMDA Receptors
- (2011) S. Li et al. JOURNAL OF NEUROSCIENCE
- Soluble amyloid -protein dimers isolated from Alzheimer cortex directly induce Tau hyperphosphorylation and neuritic degeneration
- (2011) M. Jin et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Interaction between prion protein and toxic amyloid β assemblies can be therapeutically targeted at multiple sites
- (2011) Darragh B. Freir et al. Nature Communications
- Automated 4D analysis of dendritic spine morphology: applications to stimulus-induced spine remodeling and pharmacological rescue in a disease model
- (2011) Sharon A Swanger et al. Molecular Brain
- A Oligomers Cause Localized Ca2+ Elevation, Missorting of Endogenous Tau into Dendrites, Tau Phosphorylation, and Destruction of Microtubules and Spines
- (2010) H. Zempel et al. JOURNAL OF NEUROSCIENCE
- A Mouse Model of Amyloid Oligomers: Their Contribution to Synaptic Alteration, Abnormal Tau Phosphorylation, Glial Activation, and Neuronal Loss In Vivo
- (2010) T. Tomiyama et al. JOURNAL OF NEUROSCIENCE
- -Amyloid Oligomers Induce Phosphorylation of Tau and Inactivation of Insulin Receptor Substrate via c-Jun N-Terminal Kinase Signaling: Suppression by Omega-3 Fatty Acids and Curcumin
- (2009) Q.-L. Ma et al. JOURNAL OF NEUROSCIENCE
- GluN2B subunit-containing NMDA receptor antagonists prevent A -mediated synaptic plasticity disruption in vivo
- (2009) N.-W. Hu et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- p3 peptide, a truncated form of Aβ devoid of synaptotoxic effect, does not assemble into soluble oligomers
- (2008) Fabienne Dulin et al. FEBS LETTERS
- Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory
- (2008) Ganesh M Shankar et al. NATURE MEDICINE
- Alzheimer's disease-type neuronal tau hyperphosphorylation induced by Aβ oligomers
- (2007) Fernanda G. De Felice et al. NEUROBIOLOGY OF AGING
- ADAM-10 over-expression increases cortical synaptogenesis
- (2006) Karen F.S. Bell et al. NEUROBIOLOGY OF AGING
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