Dysregulated T helper type 1 (Th1) and Th17 responses in elderly hospitalised patients with infection and sepsis

Interleukin-17A Is Associated With Alveolar Inflammation and Poor Outcomes in Acute Respiratory Distress Syndrome

(2016) Carmen Mikacenic et al.   CRITICAL CARE MEDICINE

Systemic over-release of interleukin-17 in acute kidney injury after septic shock: Clinical and experimental evidence

(2016) Panagiota Maravitsa et al.   IMMUNOLOGY LETTERS

Targeting IL-17A attenuates neonatal sepsis mortality induced by IL-18

(2016) James Lawrence Wynn et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Rapamycin attenuates acute lung injury induced by LPS through inhibition of Th17 cell proliferation in mice

(2016) Zhao Yan et al.   Scientific Reports

Inflammation-Induced Changes in Circulating T-Cell Subsets and Cytokine Production During Human Endotoxemia

(2016) Andreas Ronit et al.   JOURNAL OF INTENSIVE CARE MEDICINE

Defective cytokine production early after multiple traumas: Modulation in severe sepsis

(2015) Michael D. Paraschos et al.   CYTOKINE

The Dichotomous Pattern of IL-12R and IL-23R Expression Elucidates the Role of IL-12 and IL-23 in Inflammation

(2014) Gaëlle Chognard et al.   PLoS One

Elevated Expression of IL-23/IL-17 Pathway–Related Mediators Correlates With Exacerbation of Pulmonary Inflammation During Polymicrobial Sepsis

(2014) David M. Cauvi et al.   SHOCK

Reduction of Immunocompetent T Cells Followed by Prolonged Lymphopenia in Severe Sepsis in the Elderly*

(2013) Shigeaki Inoue et al.   CRITICAL CARE MEDICINE

The Th17/Treg balance is disturbed during aging

(2013) Vanessa Schmitt et al.   EXPERIMENTAL GERONTOLOGY

Immunosuppression in sepsis: a novel understanding of the disorder and a new therapeutic approach

(2013) Richard S Hotchkiss et al.   LANCET INFECTIOUS DISEASES

Age and CD161 Expression Contribute to Inter-Individual Variation in Interleukin-23 Response in CD8+ Memory Human T Cells

(2013) Hui Shen et al.   PLoS One

Genomic responses in mouse models poorly mimic human inflammatory diseases

(2013) Junhee Seok et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Decreased T-Cell Repertoire Diversity in Sepsis

(2012) Fabienne Venet et al.   CRITICAL CARE MEDICINE

IL-7 Restores Lymphocyte Functions in Septic Patients

(2012) F. Venet et al.   JOURNAL OF IMMUNOLOGY

Increased Percentages of T Helper Cells Producing IL-17 and Monocytes Expressing Markers of Alternative Activation in Patients with Sepsis

(2012) Milena Karina Colo Brunialti et al.   PLoS One

Phenotype and Functions of Natural Killer Cells in Critically-Ill Septic Patients

(2012) Jean-Marie Forel et al.   PLoS One

Human Monocytes Differentiate into Dendritic Cells Subsets that Induce Anergic and Regulatory T Cells in Sepsis

(2012) Valérie Faivre et al.   PLoS One

Innate Immune Responses to Systemic Acinetobacter baumannii Infection in Mice: Neutrophils, but Not Interleukin-17, Mediate Host Resistance

(2011) Jessica M. Breslow et al.   INFECTION AND IMMUNITY

Profound and persistent decrease of circulating dendritic cells is associated with ICU-acquired infection in patients with septic shock

(2011) D. Grimaldi et al.   INTENSIVE CARE MEDICINE

Chronic Mucocutaneous Candidiasis in Humans with Inborn Errors of Interleukin-17 Immunity

(2011) A. Puel et al.   SCIENCE

Th1 and Th17 cells

(2010) Jesse M. Damsker et al.   Annals of the New York Academy of Sciences

Differentiation of Effector CD4 T Cell Populations

(2010) Jinfang Zhu et al.   Annual Review of Immunology

IL-17 is a potent synergistic factor with GM-CSF in mice in stimulating myelopoiesis, dendritic cell expansion, proliferation, and functional enhancement

(2010) Bainan Liu et al.   EXPERIMENTAL HEMATOLOGY

Human interleukin 17–producing cells originate from a CD161+CD4+T cell precursor

(2008) Lorenzo Cosmi et al.   JOURNAL OF EXPERIMENTAL MEDICINE

Impaired TH17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome

(2008) Joshua D. Milner et al.   NATURE