TXNIP induced by MondoA, rather than ChREBP, suppresses cervical cancer cell proliferation, migration and invasion
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Title
TXNIP induced by MondoA, rather than ChREBP, suppresses cervical cancer cell proliferation, migration and invasion
Authors
Keywords
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Journal
JOURNAL OF BIOCHEMISTRY
Volume -, Issue -, Pages -
Publisher
Oxford University Press (OUP)
Online
2019-11-27
DOI
10.1093/jb/mvz105
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Note: Only part of the references are listed.- Cervical cancer
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- Interference of the long noncoding RNA CDKN2B‐AS1 upregulates miR‐181a‐5p/TGFβI axis to restrain the metastasis and promote apoptosis and senescence of cervical cancer cells
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- AMPK-Dependent Degradation of TXNIP upon Energy Stress Leads to Enhanced Glucose Uptake via GLUT1
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- Coordination of Nutrient Availability and Utilization by MAX- and MLX-Centered Transcription Networks
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- Lactic Acidosis Triggers Starvation Response with Paradoxical Induction of TXNIP through MondoA
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- Tandem ChoRE and CCAAT Motifs and Associated Factors Regulate Txnip Expression in Response to Glucose or Adenosine-Containing Molecules
- (2009) Fa-Xing Yu et al. PLoS One
- The glucose-responsive transcription factor ChREBP contributes to glucose-dependent anabolic synthesis and cell proliferation
- (2009) X. Tong et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Glutamine-dependent anapleurosis dictates glucose uptake and cell growth by regulating MondoA transcriptional activity
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